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The American Review of Respiratory Disease|March 1, 1986
A genetically engineered, mutant human alpha-1-proteinase inhibitor is more resistant than the normal inhibitor to oxidative inactivation by chemicals, enzymes, cells, and cigarette smokeA Janoff, C George-Nascimento, S RosenbergScience (New York, N.Y.)|August 13, 1982
Cigarette smoke contains anticoagulants against fibrin aggregation and factor XIIIa in plasmaD K Galanakis, P Laurent, A JanoffClinical Immunology and Immunopathology|August 1, 1987
Characterization of the third component of complement (C3) after activation by cigarette smokeR R Kew, B Ghebrehiwet, A JanoffClinical Immunology and Immunopathology|April 1, 1987
The fifth component of complement (C5) is necessary for maximal pulmonary leukocytosis in mice chronically exposed to cigarette smokeR R Kew, B Ghebrehiwet, A JanoffThe American Journal of Pathology|August 1, 1975
The course and nature of acinar cell death following pancreatic ligation in the guinea pigJ D Zeligs, A Janoff, A E DumontJournal of Leukocyte Biology|June 1, 1986
Inactivation of alveolar macrophage transglutaminase by oxidants in cigarette smokeW J Roth, S I Chung, A JanoffBiophysical Journal|January 5, 2000
A differential scanning calorimetry study of phosphocholines mixed with paclitaxel and its bromoacylated taxanesS Ali, S Minchey, A Janoff, et al.The American Review of Respiratory Disease|February 1, 1983
The role of oxidative processes in emphysemaA Janoff, H Carp, P Laurent, et al.Experimental Lung Research|January 1, 1988
Inhibition of human neutrophil elastase by bacterial polyanionsM Vered, S Simon, R Dearing, et al.The Biochemical Journal|May 1, 1981
The specificity of macrophage elastase on the insulin B-chainC Kettner, E Shaw, R White, et al.Pageof 7