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Annals of Internal Medicine|September 15, 1993
Diastolic compliance of hypertrophied ventricle is not acutely altered by pharmacologic agents influencing active processesD A Kass, M R Wolff, C T Ting, et al.
Archives of Ophthalmology (Chicago, Ill. : 1960)|September 15, 2005
Normal visual field test results following glaucomatous visual field end points in the Ocular Hypertension Treatment StudyJohn L Keltner, Chris A Johnson, Richard A Levine, et al.
Circulation Research|March 21, 2009
Biochemical and mechanical dysfunction in a mouse model of desmin-related myopathyAlina Maloyan, Hanna Osinska, Jan Lammerding, et al.
Circulation Research|January 23, 2014
Hyperactive adverse mechanical stress responses in dystrophic heart are coupled to transient receptor potential canonical 6 and blocked by cGMP-protein kinase G modulationKinya Seo, Peter P Rainer, Dong-Ik Lee, et al.
Eye (London, England)|February 14, 2024
Visual field progression patterns in the ocular hypertension treatment study correspond to vulnerability regions of the discAri Leshno, Nikhil Bommakanti, Carlos Gustavo De Moraes, et al.
The American Journal of Physiology|May 12, 1998
In vivo murine left ventricular pressure-volume relations by miniaturized conductance micromanometryD Georgakopoulos, W A Mitzner, C H Chen, et al.
JAMA Ophthalmology|June 5, 2020
Evaluation of a Primary Open-Angle Glaucoma Prediction Model Using Long-term Intraocular Pressure Variability Data: A Secondary Analysis of 2 Randomized Clinical TrialsMae O Gordon, Feng Gao, Julia Beiser Huecker, et al.
ASAIO Journal (American Society for Artificial Internal Organs : 1992)|July 1, 1995
Acute systolic and diastolic indices of left ventricular function after cardiomyoplasty in a chronic model of heart failureD J Polidori, E B Lankford, T Plappert, et al.
Circulation Research|January 19, 2012
Subcellular structures and function of myocytes impaired during heart failure are restored by cardiac resynchronization therapyFrank B Sachse, Natalia S Torres, Eleonora Savio-Galimberti, et al.
The Journal of Clinical Investigation|March 13, 1999
Preservation of canine myocardial high-energy phosphates during low-flow ischemia with modification of hemoglobin-oxygen affinityR G Weiss, M A Mejia, D A Kass, et al.
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