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American Journal of Physiology. Heart and Circulatory Physiology|August 29, 2006
Heart failure-associated alterations in troponin I phosphorylation impair ventricular relaxation-afterload and force-frequency responses and systolic functionKenneth C Bilchick, Jennifer G Duncan, Rajashree Ravi, et al.Circulation. Heart Failure|September 14, 2017
Heart Failure-Related Hyperphosphorylation in the Cardiac Troponin I C Terminus Has Divergent Effects on Cardiac Function In VivoYuejin Li, Guangshuo Zhu, Nazareno Paolocci, et al.Scientific Reports|November 19, 2025
Macrophages lacking TSC2 have mTORC1-dependent increased GPNMB and ameliorate ventricular dysfunction/remodeling after ischemia-reperfusionMohammad Keykhaei, Navid Koleini, Mariam Meddeb, et al.Circulation Research|December 14, 2004
Calcitonin gene-related peptide in vivo positive inotropy is attributable to regional sympatho-stimulation and is blunted in congestive heart failureTatsuo Katori, Donald B Hoover, Jeffrey L Ardell, et al.Cellular Signalling|September 16, 2008
Expression, activity, and pro-hypertrophic effects of PDE5A in cardiac myocytesManling Zhang, Norimichi Koitabashi, Takahiro Nagayama, et al.Circulation. Heart Failure|October 20, 2015
Molecular Screen Identifies Cardiac Myosin-Binding Protein-C as a Protein Kinase G-Iα SubstrateRobrecht Thoonen, Shewit Giovanni, Suresh Govindan, et al.IEEE Transactions on Medical Imaging|November 10, 2001
Enhancement of contrast echocardiography by image variability analysisB J Fetics, E Y Wong, T Murabayashi, et al.Hypertension (Dallas, Tex. : 1979)|December 3, 2014
Soluble guanylate cyclase is required for systemic vasodilation but not positive inotropy induced by nitroxyl in the mouseGuangshuo Zhu, Dieter Groneberg, Gautam Sikka, et al.AIDS (London, England)|February 4, 2012
Diastolic dysfunction is associated with myocardial viral load in simian immunodeficiency virus-infected macaquesKathleen M Kelly, Patrick M Tarwater, Jami M Karper, et al.JCI Insight|August 10, 2018
Marked disparity of microRNA modulation by cGMP-selective PDE5 versus PDE9 inhibitors in heart diseaseKristen M Kokkonen-Simon, Amir Saberi, Taishi Nakamura, et al.Pageof 61