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The Journal of Physiology|May 23, 1998
Functional expression of the Ile693Thr Na+ channel mutation associated with paramyotonia congenita in a human cell lineE Plassart-Schiess, L Lhuillier, A L George, et al.Neuron|February 23, 1999
Voltage sensors in domains III and IV, but not I and II, are immobilized by Na+ channel fast inactivationA Cha, P C Ruben, A L George, et al.Circulation Research|July 8, 2000
Activation of protein kinase A modulates trafficking of the human cardiac sodium channel in Xenopus oocytesJ Zhou, J Yi, N Hu, et al.Brain : a Journal of Neurology|June 4, 1999
Mutant channels contribute <50% to Na+ current in paramyotonia congenita muscleN Mitrovic, A L George, R Rüdel, et al.Circulation Research|October 13, 2000
Enhanced Na(+) channel intermediate inactivation in Brugada syndromeD W Wang, N Makita, A Kitabatake, et al.The Journal of Clinical Investigation|April 1, 1997
Pharmacological targeting of long QT mutant sodium channelsD W Wang, K Yazawa, N Makita, et al.The Journal of General Physiology|January 1, 1997
Subunit stoichiometry of human muscle chloride channelsC Fahlke, T Knittle, C A Gurnett, et al.Biophysical Journal|October 23, 1997
Defective fast inactivation recovery and deactivation account for sodium channel myotonia in the I1160V mutantJ E Richmond, D VanDeCarr, D E Featherstone, et al.Nature|December 11, 1997
Pore-forming segments in voltage-gated chloride channelsC Fahlke, H T Yu, C L Beck, et al.The Journal of Physiology|August 15, 1995
Different effects on gating of three myotonia-causing mutations in the inactivation gate of the human muscle sodium channelN Mitrović, A L George, H Lerche, et al.Pageof 12