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Neuron|February 1, 1994
Sodium channel mutations in paramyotonia congenita uncouple inactivation from activationM Chahine, A L George, M Zhou, et al.The Journal of Biological Chemistry|May 29, 1999
Cloning and characterization of KCC3 and KCC4, new members of the cation-chloride cotransporter gene familyD B Mount, A Mercado, L Song, et al.Proceedings of the National Academy of Sciences of the United States of America|September 1, 1989
Functional expression of the amiloride-sensitive sodium channel in Xenopus oocytesA L George, O Staub, K Geering, et al.Circulation|January 5, 2000
Cardiac Na(+) channel dysfunction in Brugada syndrome is aggravated by beta(1)-subunitN Makita, N Shirai, D W Wang, et al.FEBS Letters|August 19, 1999
A missense mutation in canine C1C-1 causes recessive myotonia congenita in the dogT H Rhodes, C H Vite, U Giger, et al.Brain : a Journal of Neurology|May 17, 2001
Skeletal muscle disuse induces fibre type-dependent enhancement of Na(+) channel expressionJ F Desaphy, S Pierno, C Léoty, et al.Neurology|November 28, 2001
Gating of myotonic Na channel mutants defines the response to mexiletine and a potent derivativeJ F Desaphy, A De Luca, P Tortorella, et al.Proceedings of the National Academy of Sciences of the United States of America|December 20, 1994
Sodium channel mutations in paramyotonia congenita exhibit similar biophysical phenotypes in vitroN Yang, S Ji, M Zhou, et al.Circulation|June 22, 1999
Congenital long-QT syndrome caused by a novel mutation in a conserved acidic domain of the cardiac Na+ channelJ Wei, D W Wang, M Alings, et al.Circulation|September 6, 2001
Gating-dependent mechanisms for flecainide action in SCN5A-linked arrhythmia syndromesP C Viswanathan, C R Bezzina, A L George, et al.Pageof 12