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A L Joyner

Showing results (81-90 of 93) with videos related to

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Mechanisms of Development|October 19, 2002
A novel gene, GliH1, with homology to the Gli zinc finger domain not required for mouse developmentM Nakashima, N Tanese, M Ito, et al.
Nature Medicine|November 1, 1996
Glucose intolerance but normal satiety in mice with a null mutation in the glucagon-like peptide 1 receptor geneL A Scrocchi, T J Brown, N MaClusky, et al.
Blood|August 29, 2000
LSP1 modulates leukocyte populations in resting and inflamed peritoneumJ Jongstra-Bilen, V L Misener, C Wang, et al.
Nature Genetics|December 17, 1997
Huntingtin is required for neurogenesis and is not impaired by the Huntington's disease CAG expansionJ K White, W Auerbach, M P Duyao, et al.
Development (Cambridge, England)|March 22, 2000
Mouse Gli1 mutants are viable but have defects in SHH signaling in combination with a Gli2 mutationH L Park, C Bai, K A Platt, et al.
Human Molecular Genetics|November 16, 2001
The HD mutation causes progressive lethal neurological disease in mice expressing reduced levels of huntingtinW Auerbach, M S Hurlbert, P Hilditch-Maguire, et al.
Nature Genetics|March 1, 1995
Genomic imprinting of Mash2, a mouse gene required for trophoblast developmentF Guillemot, T Caspary, S M Tilghman, et al.
Cold Spring Harbor Symposia on Quantitative Biology|January 1, 1996
Targeted inactivation of the mouse Huntington's disease gene homolog HdhM E MacDonald, M Duyao, T Calzonetti, et al.
Science (New York, N.Y.)|July 21, 1995
Inactivation of the mouse Huntington's disease gene homolog HdhM P Duyao, A B Auerbach, A Ryan, et al.
Human Molecular Genetics|January 15, 1999
Length-dependent gametic CAG repeat instability in the Huntington's disease knock-in mouseV C Wheeler, W Auerbach, J K White, et al.
Pageof 10

Showing results (81-90 of 93) with videos related to

Sort By:
Pageof 10
Mechanisms of Development|October 19, 2002
A novel gene, GliH1, with homology to the Gli zinc finger domain not required for mouse developmentM Nakashima, N Tanese, M Ito, et al.
Nature Medicine|November 1, 1996
Glucose intolerance but normal satiety in mice with a null mutation in the glucagon-like peptide 1 receptor geneL A Scrocchi, T J Brown, N MaClusky, et al.
Blood|August 29, 2000
LSP1 modulates leukocyte populations in resting and inflamed peritoneumJ Jongstra-Bilen, V L Misener, C Wang, et al.
Nature Genetics|December 17, 1997
Huntingtin is required for neurogenesis and is not impaired by the Huntington's disease CAG expansionJ K White, W Auerbach, M P Duyao, et al.
Development (Cambridge, England)|March 22, 2000
Mouse Gli1 mutants are viable but have defects in SHH signaling in combination with a Gli2 mutationH L Park, C Bai, K A Platt, et al.
Human Molecular Genetics|November 16, 2001
The HD mutation causes progressive lethal neurological disease in mice expressing reduced levels of huntingtinW Auerbach, M S Hurlbert, P Hilditch-Maguire, et al.
Nature Genetics|March 1, 1995
Genomic imprinting of Mash2, a mouse gene required for trophoblast developmentF Guillemot, T Caspary, S M Tilghman, et al.
Cold Spring Harbor Symposia on Quantitative Biology|January 1, 1996
Targeted inactivation of the mouse Huntington's disease gene homolog HdhM E MacDonald, M Duyao, T Calzonetti, et al.
Science (New York, N.Y.)|July 21, 1995
Inactivation of the mouse Huntington's disease gene homolog HdhM P Duyao, A B Auerbach, A Ryan, et al.
Human Molecular Genetics|January 15, 1999
Length-dependent gametic CAG repeat instability in the Huntington's disease knock-in mouseV C Wheeler, W Auerbach, J K White, et al.
Pageof 10