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A Lanuza

Showing results (51-60 of 87) with videos related to

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Cells|December 11, 2019
The Impact of Kinases in Amyotrophic Lateral Sclerosis at the Neuromuscular Synapse: Insights into BDNF/TrkB and PKC SignalingMaria A Lanuza, Laia Just-Borràs, Erica Hurtado, et al.
Cells|October 27, 2019
Opposed Actions of PKA Isozymes (RI and RII) and PKC Isoforms (cPKCβI and nPKCε) in Neuromuscular Developmental Synapse EliminationNeus Garcia, Cori Balañà, Maria A Lanuza, et al.
Frontiers in Aging Neuroscience|February 24, 2017
Presynaptic Muscarinic Acetylcholine Receptors and TrkB Receptor Cooperate in the Elimination of Redundant Motor Nerve Terminals during DevelopmentLaura Nadal, Neus Garcia, Erica Hurtado, et al.
Archivos De La Sociedad Espanola De Oftalmologia|June 30, 2005
[Management of lymphoid hyperplasia of the ocular adnexa]A Lanuza García, A L López Ramos, J C Pinto Bonilla, et al.
Molecular Brain|June 25, 2016
Presynaptic muscarinic acetylcholine autoreceptors (M1, M2 and M4 subtypes), adenosine receptors (A1 and A2A) and tropomyosin-related kinase B receptor (TrkB) modulate the developmental synapse elimination process at the neuromuscular junctionLaura Nadal, Neus Garcia, Erica Hurtado, et al.
Journal of Neuroscience Research|March 14, 2002
Pre- and postsynaptic maturation of the neuromuscular junction during neonatal synapse elimination depends on protein kinase CMaria A Lanuza, Neus Garcia, Manel Santafé, et al.
Journal of the Peripheral Nervous System : JPNS|September 14, 2012
Exogenous ciliary neurotrophic factor (CNTF) reduces synaptic depression during repetitive stimulationNeus Garcia, Manel M Santafé, Marta Tomàs, et al.
Neuroscience Letters|October 19, 2013
Cellular localization of the atypical isoforms of protein kinase C (aPKCζ/PKMζ and aPKCλ/ι) on the neuromuscular synapseNúria Besalduch, Maria A Lanuza, Neus Garcia, et al.
Archivos De La Sociedad Espanola De Oftalmologia|July 22, 2024
Ocular ischemia syndrome after aesthetic treatment with hyaluronic acidE Gracia Rovira, S Natividad Andreu, N Montalt Barrachina, et al.
Frontiers in Molecular Neuroscience|August 30, 2017
Membrane Receptor-Induced Changes of the Protein Kinases A and C Activity May Play a Leading Role in Promoting Developmental Synapse Elimination at the Neuromuscular JunctionJosep M Tomàs, Neus Garcia, Maria A Lanuza, et al.
Pageof 9

Showing results (51-60 of 87) with videos related to

Sort By:
Pageof 9
Cells|December 11, 2019
The Impact of Kinases in Amyotrophic Lateral Sclerosis at the Neuromuscular Synapse: Insights into BDNF/TrkB and PKC SignalingMaria A Lanuza, Laia Just-Borràs, Erica Hurtado, et al.
Cells|October 27, 2019
Opposed Actions of PKA Isozymes (RI and RII) and PKC Isoforms (cPKCβI and nPKCε) in Neuromuscular Developmental Synapse EliminationNeus Garcia, Cori Balañà, Maria A Lanuza, et al.
Frontiers in Aging Neuroscience|February 24, 2017
Presynaptic Muscarinic Acetylcholine Receptors and TrkB Receptor Cooperate in the Elimination of Redundant Motor Nerve Terminals during DevelopmentLaura Nadal, Neus Garcia, Erica Hurtado, et al.
Archivos De La Sociedad Espanola De Oftalmologia|June 30, 2005
[Management of lymphoid hyperplasia of the ocular adnexa]A Lanuza García, A L López Ramos, J C Pinto Bonilla, et al.
Molecular Brain|June 25, 2016
Presynaptic muscarinic acetylcholine autoreceptors (M1, M2 and M4 subtypes), adenosine receptors (A1 and A2A) and tropomyosin-related kinase B receptor (TrkB) modulate the developmental synapse elimination process at the neuromuscular junctionLaura Nadal, Neus Garcia, Erica Hurtado, et al.
Journal of Neuroscience Research|March 14, 2002
Pre- and postsynaptic maturation of the neuromuscular junction during neonatal synapse elimination depends on protein kinase CMaria A Lanuza, Neus Garcia, Manel Santafé, et al.
Journal of the Peripheral Nervous System : JPNS|September 14, 2012
Exogenous ciliary neurotrophic factor (CNTF) reduces synaptic depression during repetitive stimulationNeus Garcia, Manel M Santafé, Marta Tomàs, et al.
Neuroscience Letters|October 19, 2013
Cellular localization of the atypical isoforms of protein kinase C (aPKCζ/PKMζ and aPKCλ/ι) on the neuromuscular synapseNúria Besalduch, Maria A Lanuza, Neus Garcia, et al.
Archivos De La Sociedad Espanola De Oftalmologia|July 22, 2024
Ocular ischemia syndrome after aesthetic treatment with hyaluronic acidE Gracia Rovira, S Natividad Andreu, N Montalt Barrachina, et al.
Frontiers in Molecular Neuroscience|August 30, 2017
Membrane Receptor-Induced Changes of the Protein Kinases A and C Activity May Play a Leading Role in Promoting Developmental Synapse Elimination at the Neuromuscular JunctionJosep M Tomàs, Neus Garcia, Maria A Lanuza, et al.
Pageof 9