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A Marschner

Showing results (61-70 of 71) with videos related to

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Bioscience Reports|October 22, 2017
Gene expression profiling of the Notch-AhR-IL22 axis at homeostasis and in response to tissue injuryMarc Weidenbusch, Severin Rodler, Shangqing Song, et al.
Kidney International|December 16, 2017
The macrophage phenotype and inflammasome component NLRP3 contributes to nephrocalcinosis-related chronic kidney disease independent from IL-1-mediated tissue injuryHans-Joachim Anders, Beatriz Suarez-Alvarez, Melissa Grigorescu, et al.
Journal of the American Society of Nephrology : JASN|March 1, 2023
Tubular Epithelial Cell HMGB1 Promotes AKI-CKD Transition by Sensitizing Cycling Tubular Cells to Oxidative Stress: A Rationale for Targeting HMGB1 during AKI RecoveryZhi Bo Zhao, Julian A Marschner, Takamasa Iwakura, et al.
Frontiers in Immunology|September 25, 2023
No NLRP3 inflammasome activity in kidney epithelial cells, not even when the NLRP3-A350V Muckle-Wells variant is expressed in podocytes of diabetic miceSophie Carina Kunte, Julian A Marschner, Martin Klaus, et al.
Journal of Medicinal Chemistry|July 31, 2024
Structural Optimization of Oxaprozin for Selective Inverse Nurr1 AgonismSabine Willems, Romy Busch, Felix Nawa, et al.
Journal of the American Society of Nephrology : JASN|January 15, 2020
The Kidney Contains Ontogenetically Distinct Dendritic Cell and Macrophage Subtypes throughout Development That Differ in Their Inflammatory PropertiesNatallia Salei, Stephan Rambichler, Johanna Salvermoser, et al.
Journal of the American Society of Nephrology : JASN|May 7, 2016
Intestinal Dysbiosis, Barrier Dysfunction, and Bacterial Translocation Account for CKD-Related Systemic InflammationKirstin Andersen, Marie Sophie Kesper, Julian A Marschner, et al.
Journal of the American Society of Nephrology : JASN|July 13, 2019
Mitochondria Permeability Transition versus Necroptosis in Oxalate-Induced AKIShrikant Ramesh Mulay, Mohsen M Honarpisheh, Orestes Foresto-Neto, et al.
Journal of the American Society of Nephrology : JASN|September 11, 2016
Hyperoxaluria Requires TNF Receptors to Initiate Crystal Adhesion and Kidney Stone DiseaseShrikant R Mulay, Jonathan N Eberhard, Jyaysi Desai, et al.
Nature Communications|April 11, 2018
Endocycle-related tubular cell hypertrophy and progenitor proliferation recover renal function after acute kidney injuryElena Lazzeri, Maria Lucia Angelotti, Anna Peired, et al.
Pageof 8

Showing results (61-70 of 71) with videos related to

Sort By:
Pageof 8
Bioscience Reports|October 22, 2017
Gene expression profiling of the Notch-AhR-IL22 axis at homeostasis and in response to tissue injuryMarc Weidenbusch, Severin Rodler, Shangqing Song, et al.
Kidney International|December 16, 2017
The macrophage phenotype and inflammasome component NLRP3 contributes to nephrocalcinosis-related chronic kidney disease independent from IL-1-mediated tissue injuryHans-Joachim Anders, Beatriz Suarez-Alvarez, Melissa Grigorescu, et al.
Journal of the American Society of Nephrology : JASN|March 1, 2023
Tubular Epithelial Cell HMGB1 Promotes AKI-CKD Transition by Sensitizing Cycling Tubular Cells to Oxidative Stress: A Rationale for Targeting HMGB1 during AKI RecoveryZhi Bo Zhao, Julian A Marschner, Takamasa Iwakura, et al.
Frontiers in Immunology|September 25, 2023
No NLRP3 inflammasome activity in kidney epithelial cells, not even when the NLRP3-A350V Muckle-Wells variant is expressed in podocytes of diabetic miceSophie Carina Kunte, Julian A Marschner, Martin Klaus, et al.
Journal of Medicinal Chemistry|July 31, 2024
Structural Optimization of Oxaprozin for Selective Inverse Nurr1 AgonismSabine Willems, Romy Busch, Felix Nawa, et al.
Journal of the American Society of Nephrology : JASN|January 15, 2020
The Kidney Contains Ontogenetically Distinct Dendritic Cell and Macrophage Subtypes throughout Development That Differ in Their Inflammatory PropertiesNatallia Salei, Stephan Rambichler, Johanna Salvermoser, et al.
Journal of the American Society of Nephrology : JASN|May 7, 2016
Intestinal Dysbiosis, Barrier Dysfunction, and Bacterial Translocation Account for CKD-Related Systemic InflammationKirstin Andersen, Marie Sophie Kesper, Julian A Marschner, et al.
Journal of the American Society of Nephrology : JASN|July 13, 2019
Mitochondria Permeability Transition versus Necroptosis in Oxalate-Induced AKIShrikant Ramesh Mulay, Mohsen M Honarpisheh, Orestes Foresto-Neto, et al.
Journal of the American Society of Nephrology : JASN|September 11, 2016
Hyperoxaluria Requires TNF Receptors to Initiate Crystal Adhesion and Kidney Stone DiseaseShrikant R Mulay, Jonathan N Eberhard, Jyaysi Desai, et al.
Nature Communications|April 11, 2018
Endocycle-related tubular cell hypertrophy and progenitor proliferation recover renal function after acute kidney injuryElena Lazzeri, Maria Lucia Angelotti, Anna Peired, et al.
Pageof 8