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The Journal of Cell Biology|September 1, 1995
Increasing neurofilament subunit NF-M expression reduces axonal NF-H, inhibits radial growth, and results in neurofilamentous accumulation in motor neuronsP C Wong, J Marszalek, T O Crawford, et al.Molecular and Cellular Biology|February 1, 1990
A rapidly rearranging retrotransposon within the miniexon gene locus of Crithidia fasciculataA Gabriel, T J Yen, D C Schwartz, et al.Proceedings of the National Academy of Sciences of the United States of America|August 5, 1998
Absence of neurofilaments reduces the selective vulnerability of motor neurons and slows disease caused by a familial amyotrophic lateral sclerosis-linked superoxide dismutase 1 mutantT L Williamson, L I Bruijn, Q Zhu, et al.The EMBO Journal|May 1, 1991
CENP-E, a novel human centromere-associated protein required for progression from metaphase to anaphaseT J Yen, D A Compton, D Wise, et al.Proceedings of the National Academy of Sciences of the United States of America|July 8, 1997
Elevated free nitrotyrosine levels, but not protein-bound nitrotyrosine or hydroxyl radicals, throughout amyotrophic lateral sclerosis (ALS)-like disease implicate tyrosine nitration as an aberrant in vivo property of one familial ALS-linked superoxide dismutase 1 mutantL I Bruijn, M F Beal, M W Becher, et al.Amyotrophic Lateral Sclerosis and Other Motor Neuron Disorders : Official Publication of the World Federation of Neurology, Research Group on Motor Neuron Diseases|July 24, 2001
New consensus research on neuropathological aspects of familial amyotrophic lateral sclerosis with superoxide dismutase 1 (SOD1) gene mutations: inclusions containing SOD1 in neurons and astrocytesS Kato, M Takikawa, K Nakashima, et al.Cell|July 20, 2001
Mps1 is a kinetochore-associated kinase essential for the vertebrate mitotic checkpointA Abrieu, L Magnaghi-Jaulin, J A Kahana, et al.Proceedings of the National Academy of Sciences of the United States of America|August 19, 1997
CENP-E is an essential kinetochore motor in maturing oocytes and is masked during mos-dependent, cell cycle arrest at metaphase IIN S Duesbery, T Choi, K D Brown, et al.Annals of the New York Academy of Sciences|February 24, 2001
The value of transgenic models for the study of neurodegenerative diseasesD L Price, P C Wong, A L Markowska, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|November 1, 1994
Regional modulation of neurofilament organization by myelination in normal axonsS T Hsieh, G J Kidd, T O Crawford, et al.Pageof 15