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Journal of Neurochemistry|January 14, 1999
The copper chaperone CCS is abundant in neurons and astrocytes in human and rodent brainJ D Rothstein, M Dykes-Hoberg, L B Corson, et al.
The Journal of Cell Biology|November 1, 1996
Neurofilament subunit NF-H modulates axonal diameter by selectively slowing neurofilament transportJ R Marszalek, T L Williamson, M K Lee, et al.
The EMBO Journal|January 1, 1991
Mammalian nuclei contain foci which are highly enriched in components of the pre-mRNA splicing machineryM Carmo-Fonseca, D Tollervey, R Pepperkok, et al.
Neurology|April 14, 2004
Alzheimer disease without neocortical neurofibrillary tangles: "a second look"P Tiraboschi, M N Sabbagh, L A Hansen, et al.
The Journal of Cell Biology|April 1, 1987
Molecular cloning of cDNA for CENP-B, the major human centromere autoantigenW C Earnshaw, K F Sullivan, P S Machlin, et al.
Science (New York, N.Y.)|September 22, 1998
Aggregation and motor neuron toxicity of an ALS-linked SOD1 mutant independent from wild-type SOD1L I Bruijn, M K Houseweart, S Kato, et al.
Acta Neuropathologica|October 25, 2000
Advanced glycation endproduct-modified superoxide dismutase-1 (SOD1)-positive inclusions are common to familial amyotrophic lateral sclerosis patients with SOD1 gene mutations and transgenic mice expressing human SOD1 with a G85R mutationS Kato, S Horiuchi, J Liu, et al.
The Journal of Cell Biology|June 1, 1996
Subunit composition of neurofilaments specifies axonal diameterZ Xu, J R Marszalek, M K Lee, et al.
The Journal of Biological Chemistry|February 17, 1995
Superoxide dismutase 1 subunits with mutations linked to familial amyotrophic lateral sclerosis do not affect wild-type subunit functionD R Borchelt, M Guarnieri, P C Wong, et al.
Neuron|June 1, 1995
An adverse property of a familial ALS-linked SOD1 mutation causes motor neuron disease characterized by vacuolar degeneration of mitochondriaP C Wong, C A Pardo, D R Borchelt, et al.
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