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American Journal of Human Genetics|February 7, 2008
Three genome-wide association studies and a linkage analysis identify HERC2 as a human iris color geneManfred Kayser, Fan Liu, A Cecile J W Janssens, et al.
Obesity (Silver Spring, Md.)|October 24, 2009
Linkage and genome-wide association analysis of obesity-related phenotypes: association of weight with the MGAT1 geneAsa Johansson, Fabio Marroni, Caroline Hayward, et al.
Journal of Medical Genetics|November 8, 2005
The G6055A (G2019S) mutation in LRRK2 is frequent in both early and late onset Parkinson's disease and originates from a common ancestorS Goldwurm, A Di Fonzo, E J Simons, et al.
European Journal of Human Genetics : EJHG|January 17, 2013
Meta-analysis of telomere length in 19,713 subjects reveals high heritability, stronger maternal inheritance and a paternal age effectLinda Broer, Veryan Codd, Dale R Nyholt, et al.
Molecular Psychiatry|November 23, 2011
A K(ATP) channel gene effect on sleep duration: from genome-wide association studies to function in DrosophilaK V Allebrandt, N Amin, B Müller-Myhsok, et al.
Plos One|October 21, 2014
The challenges of genome-wide interaction studies: lessons to learn from the analysis of HDL blood levelsElisabeth M van Leeuwen, Françoise A S Smouter, Tony Kam-Thong, et al.
Human Molecular Genetics|July 23, 2013
Genetic influences on plasma CFH and CFHR1 concentrations and their role in susceptibility to age-related macular degenerationMorad Ansari, Paul M McKeigue, Christine Skerka, et al.
BMC Medical Genetics|March 13, 2010
A meta-analysis of genome-wide data from five European isolates reveals an association of COL22A1, SYT1, and GABRR2 with serum creatinine levelCristian Pattaro, Alessandro De Grandi, Veronique Vitart, et al.
Movement Disorders : Official Journal of the Movement Disorder Society|December 8, 2004
Novel parkin mutations detected in patients with early-onset Parkinson's diseaseAida M Bertoli-Avella, José L Giroud-Benitez, Ali Akyol, et al.
European Journal of Medical Genetics|March 25, 2009
Extending the phenotype of recurrent rearrangements of 16p11.2: deletions in mentally retarded patients without autism and in normal individualsE K Bijlsma, A C J Gijsbers, J H M Schuurs-Hoeijmakers, et al.
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