A P Cope|F M Brennan - JoVE Visualize

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European Journal of Immunology|August 1, 1994

Lymphotoxin acts as an autocrine growth factor for Epstein-Barr virus-transformed B cells and differentiated Burkitt lymphoma cell linesD L Gibbons, M Rowe, A P Cope, et al.

European Journal of Immunology|July 1, 1992

Enhanced expression of tumor necrosis factor receptor mRNA and protein in mononuclear cells isolated from rheumatoid arthritis synovial jointsF M Brennan, D L Gibbons, T Mitchell, et al.

Scandinavian Journal of Immunology|July 1, 1995

TNF inhibitors are produced spontaneously by rheumatoid and osteoarthritic synovial joint cell cultures: evidence of feedback control of TNF actionF M Brennan, D L Gibbons, A P Cope, et al.

Immunology|January 1, 1995

Soluble TNF receptor production by activated T lymphocytes: differential effects of acute and chronic exposure to TNFA P Cope, D Aderka, D Wallach, et al.

Clinical and Experimental Rheumatology|March 1, 1993

TNF-alpha in rheumatoid arthritis and prospects of anti-TNF therapyR N Maini, F M Brennan, R Williams, et al.

Progress in Growth Factor Research|January 1, 1992

Evaluation of the role of cytokines in autoimmune disease: the importance of TNF alpha in rheumatoid arthritisM Feldmann, F M Brennan, R O Williams, et al.

Cytokine|May 1, 1993

Differential regulation of tumour necrosis factor receptors (TNF-R) by IL-4; upregulation of P55 and P75 TNF-R on synovial joint mononuclear cellsA P Cope, D L Gibbons, D Aderka, et al.

Rheumatology (Oxford, England)|May 6, 2003

Exploring the reciprocal relationship between immunity and inflammation in chronic inflammatory arthritisA P Cope

Current Opinion in Immunology|January 23, 1999

Regulation of autoimmunity by proinflammatory cytokinesA P Cope

The Journal of Clinical Investigation|August 1, 1994

Chronic exposure to tumor necrosis factor (TNF) in vitro impairs the activation of T cells through the T cell receptor/CD3 complex; reversal in vivo by anti-TNF antibodies in patients with rheumatoid arthritisA P Cope, M Londei, N R Chu, et al.

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Showing results (1-10 of 119) with videos related to

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Pageof 12

European Journal of Immunology|August 1, 1994

Lymphotoxin acts as an autocrine growth factor for Epstein-Barr virus-transformed B cells and differentiated Burkitt lymphoma cell linesD L Gibbons, M Rowe, A P Cope, et al.

European Journal of Immunology|July 1, 1992

Enhanced expression of tumor necrosis factor receptor mRNA and protein in mononuclear cells isolated from rheumatoid arthritis synovial jointsF M Brennan, D L Gibbons, T Mitchell, et al.

Scandinavian Journal of Immunology|July 1, 1995

TNF inhibitors are produced spontaneously by rheumatoid and osteoarthritic synovial joint cell cultures: evidence of feedback control of TNF actionF M Brennan, D L Gibbons, A P Cope, et al.

Immunology|January 1, 1995

Soluble TNF receptor production by activated T lymphocytes: differential effects of acute and chronic exposure to TNFA P Cope, D Aderka, D Wallach, et al.

Clinical and Experimental Rheumatology|March 1, 1993

TNF-alpha in rheumatoid arthritis and prospects of anti-TNF therapyR N Maini, F M Brennan, R Williams, et al.

Progress in Growth Factor Research|January 1, 1992

Evaluation of the role of cytokines in autoimmune disease: the importance of TNF alpha in rheumatoid arthritisM Feldmann, F M Brennan, R O Williams, et al.

Cytokine|May 1, 1993

Differential regulation of tumour necrosis factor receptors (TNF-R) by IL-4; upregulation of P55 and P75 TNF-R on synovial joint mononuclear cellsA P Cope, D L Gibbons, D Aderka, et al.

Rheumatology (Oxford, England)|May 6, 2003

Exploring the reciprocal relationship between immunity and inflammation in chronic inflammatory arthritisA P Cope

Current Opinion in Immunology|January 23, 1999

Regulation of autoimmunity by proinflammatory cytokinesA P Cope

The Journal of Clinical Investigation|August 1, 1994

Chronic exposure to tumor necrosis factor (TNF) in vitro impairs the activation of T cells through the T cell receptor/CD3 complex; reversal in vivo by anti-TNF antibodies in patients with rheumatoid arthritisA P Cope, M Londei, N R Chu, et al.

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