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A Powis

Showing results (51-60 of 68) with videos related to

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Journal of Visualized Experiments : Jove|February 25, 2014
Excitotoxic stimulation of brain microslices as an in vitro model of strokeKathryn A Skelding, Jacinta M Arellano, David A Powis, et al.
Journal of Neurochemistry|August 11, 2000
Mobilizing store Ca(2+) in the presence of La(3+) evokes exocytosis in bovine chromaffin cellsP D Marley, P J Bales, M Zerbes, et al.
Neuroscience Letters|January 22, 2000
Histamine-stimulated phospholipase C activity in bovine adrenal medullary chromaffin cells: the effect of chloride-channel antagonists and low extracellular chloride concentrationsE L Roberts-Thomson, S M Palmer, D A Powis, et al.
Education for Health (Abingdon, England)|January 27, 2004
How do Australian doctors with different pre-medical school backgrounds perform as interns?M R Grey, S A Pearson, I E Rolfe, et al.
Molecular Neurobiology|January 1, 1991
Mechanisms of synaptic plasticity. Changes in postsynaptic densities and glutamate receptors in chicken forebrain during maturationJ A Rostas, J M Kavanagh, P R Dodd, et al.
Biochemical Pharmacology|February 9, 1996
Tyrosine hydroxylase phosphorylation in bovine adrenal chromaffin cells. Clonidine stimulates basal but inhibits nicotinic receptor evoked phosphorylationP R Dunkley, A Côté, S M Harrison, et al.
Cell Calcium|May 1, 1996
Mn2+ can substitute for Ca2+ in causing catecholamine secretion but not for increasing tyrosine hydroxylase phosphorylation in bovine adrenal chromaffin cellsD A Powis, K J O'Brien, S M Harrison, et al.
General Pharmacology|January 1, 1989
A-23187 evoked transmitter release from rabbit pulmonary artery and its inhibition by reactivation of sodium-pumpT L Török, D Rácz, P T Tóth, et al.
Neuropathology and Applied Neurobiology|July 22, 2014
Increased levels of UCHL1 are a compensatory response to disrupted ubiquitin homeostasis in spinal muscular atrophy and do not represent a viable therapeutic targetRachael A Powis, Chantal A Mutsaers, Thomas M Wishart, et al.
Developmental Neuroscience|October 19, 2006
Maturational changes in the subunit composition of AMPA receptors and the functional consequences of their activation in chicken forebrainPaola V Migues, Martin Cammarota, Jacinta Kavanagh, et al.
Pageof 7

Showing results (51-60 of 68) with videos related to

Sort By:
Pageof 7
Journal of Visualized Experiments : Jove|February 25, 2014
Excitotoxic stimulation of brain microslices as an in vitro model of strokeKathryn A Skelding, Jacinta M Arellano, David A Powis, et al.
Journal of Neurochemistry|August 11, 2000
Mobilizing store Ca(2+) in the presence of La(3+) evokes exocytosis in bovine chromaffin cellsP D Marley, P J Bales, M Zerbes, et al.
Neuroscience Letters|January 22, 2000
Histamine-stimulated phospholipase C activity in bovine adrenal medullary chromaffin cells: the effect of chloride-channel antagonists and low extracellular chloride concentrationsE L Roberts-Thomson, S M Palmer, D A Powis, et al.
Education for Health (Abingdon, England)|January 27, 2004
How do Australian doctors with different pre-medical school backgrounds perform as interns?M R Grey, S A Pearson, I E Rolfe, et al.
Molecular Neurobiology|January 1, 1991
Mechanisms of synaptic plasticity. Changes in postsynaptic densities and glutamate receptors in chicken forebrain during maturationJ A Rostas, J M Kavanagh, P R Dodd, et al.
Biochemical Pharmacology|February 9, 1996
Tyrosine hydroxylase phosphorylation in bovine adrenal chromaffin cells. Clonidine stimulates basal but inhibits nicotinic receptor evoked phosphorylationP R Dunkley, A Côté, S M Harrison, et al.
Cell Calcium|May 1, 1996
Mn2+ can substitute for Ca2+ in causing catecholamine secretion but not for increasing tyrosine hydroxylase phosphorylation in bovine adrenal chromaffin cellsD A Powis, K J O'Brien, S M Harrison, et al.
General Pharmacology|January 1, 1989
A-23187 evoked transmitter release from rabbit pulmonary artery and its inhibition by reactivation of sodium-pumpT L Török, D Rácz, P T Tóth, et al.
Neuropathology and Applied Neurobiology|July 22, 2014
Increased levels of UCHL1 are a compensatory response to disrupted ubiquitin homeostasis in spinal muscular atrophy and do not represent a viable therapeutic targetRachael A Powis, Chantal A Mutsaers, Thomas M Wishart, et al.
Developmental Neuroscience|October 19, 2006
Maturational changes in the subunit composition of AMPA receptors and the functional consequences of their activation in chicken forebrainPaola V Migues, Martin Cammarota, Jacinta Kavanagh, et al.
Pageof 7