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A R Giles

Showing results (61-70 of 72) with videos related to

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Seminars in Thrombosis and Hemostasis|July 1, 1990
Pathologic fibrinolysis as a cause of clinical bleedingD C Stump, F B Taylor, M E Nesheim, et al.
British Journal of Haematology|December 1, 1991
Recurring mutations at CpG dinucleotides in the region of the von Willebrand factor gene encoding the glycoprotein Ib binding domain, in patients with type IIB von Willebrand's diseaseD Lillicrap, E W Murray, K Benford, et al.
Blood|June 1, 1993
Biochemical, immunological, and in vivo functional characterization of B-domain-deleted factor VIIID D Pittman, E M Alderman, K N Tomkinson, et al.
The Journal of Biological Chemistry|September 25, 1991
The effect of plasma von Willebrand factor on the binding of human factor VIII to thrombin-activated human plateletsM Nesheim, D D Pittman, A R Giles, et al.
Thrombosis and Haemostasis|May 3, 1993
Activation of protein C and its distribution between its inhibitors, protein C inhibitor, alpha 1-antitrypsin and alpha 2-macroglobulin, in patients with disseminated intravascular coagulationM F Scully, C H Toh, H Hoogendoorn, et al.
American Journal of Clinical Pathology|October 1, 1989
Absence of a bleeding tendency in severe acquired von Willebrand's disease. The role of platelet von Willebrand factor in maintaining normal hemostasisJ Drouin, D P Lillicrap, C A Izaguirre, et al.
Blood|April 11, 1992
Anticoagulant and fibrinolytic activities are promoted, not retarded, in vivo after thrombin generation in the presence of a monoclonal antibody that inhibits activation of protein CF B Taylor, H Hoogendoorn, A C Chang, et al.
American Journal of Medical Genetics|March 1, 1987
Recombination between the factor VIII gene and the DXS52 locus gives the most probable genetic order as centromere-fra(X)-DXS15-DXS52-F8C-telomereL M Mulligan, H J Grover, V S Blanchette, et al.
Atherosclerosis|March 24, 2000
Aortic endothelial cell von Willebrand factor content, and circulating plasminogen activator inhibitor-1 are increased, but expression of endothelial leukocyte adhesion molecules is unchanged in insulin-dependent diabetic BB ratsJ C Ribau, J A Samis, Y A Senis, et al.
Clinical Genetics|July 1, 1987
Carrier detection of haemophilia A using DNA markers in families with an isolated affected maleH Grover, M A Phillips, D P Lillicrap, et al.
Pageof 8

Showing results (61-70 of 72) with videos related to

Sort By:
Pageof 8
Seminars in Thrombosis and Hemostasis|July 1, 1990
Pathologic fibrinolysis as a cause of clinical bleedingD C Stump, F B Taylor, M E Nesheim, et al.
British Journal of Haematology|December 1, 1991
Recurring mutations at CpG dinucleotides in the region of the von Willebrand factor gene encoding the glycoprotein Ib binding domain, in patients with type IIB von Willebrand's diseaseD Lillicrap, E W Murray, K Benford, et al.
Blood|June 1, 1993
Biochemical, immunological, and in vivo functional characterization of B-domain-deleted factor VIIID D Pittman, E M Alderman, K N Tomkinson, et al.
The Journal of Biological Chemistry|September 25, 1991
The effect of plasma von Willebrand factor on the binding of human factor VIII to thrombin-activated human plateletsM Nesheim, D D Pittman, A R Giles, et al.
Thrombosis and Haemostasis|May 3, 1993
Activation of protein C and its distribution between its inhibitors, protein C inhibitor, alpha 1-antitrypsin and alpha 2-macroglobulin, in patients with disseminated intravascular coagulationM F Scully, C H Toh, H Hoogendoorn, et al.
American Journal of Clinical Pathology|October 1, 1989
Absence of a bleeding tendency in severe acquired von Willebrand's disease. The role of platelet von Willebrand factor in maintaining normal hemostasisJ Drouin, D P Lillicrap, C A Izaguirre, et al.
Blood|April 11, 1992
Anticoagulant and fibrinolytic activities are promoted, not retarded, in vivo after thrombin generation in the presence of a monoclonal antibody that inhibits activation of protein CF B Taylor, H Hoogendoorn, A C Chang, et al.
American Journal of Medical Genetics|March 1, 1987
Recombination between the factor VIII gene and the DXS52 locus gives the most probable genetic order as centromere-fra(X)-DXS15-DXS52-F8C-telomereL M Mulligan, H J Grover, V S Blanchette, et al.
Atherosclerosis|March 24, 2000
Aortic endothelial cell von Willebrand factor content, and circulating plasminogen activator inhibitor-1 are increased, but expression of endothelial leukocyte adhesion molecules is unchanged in insulin-dependent diabetic BB ratsJ C Ribau, J A Samis, Y A Senis, et al.
Clinical Genetics|July 1, 1987
Carrier detection of haemophilia A using DNA markers in families with an isolated affected maleH Grover, M A Phillips, D P Lillicrap, et al.
Pageof 8