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Showing results (71-80 of 80) with videos related to

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Circulation Research|November 22, 2001
Dilated cardiomyopathy and sudden death resulting from constitutive activation of protein kinase aC L Antos, N Frey, S O Marx, et al.
Cell|May 20, 1994
Stabilization of calcium release channel (ryanodine receptor) function by FK506-binding proteinA B Brillantes, K Ondrias, A Scott, et al.
The Journal of Clinical Investigation|September 1, 1992
Expression of dihydropyridine receptor (Ca2+ channel) and calsequestrin genes in the myocardium of patients with end-stage heart failureT Takahashi, P D Allen, R V Lacro, et al.
The Journal of Biological Chemistry|June 3, 1994
Localization of the high and low affinity [3H]ryanodine binding sites on the skeletal muscle Ca2+ release channelC Callaway, A Seryshev, J P Wang, et al.
Genomics|November 1, 1991
Evidence for genetic heterogeneity in malignant hyperthermia susceptibilityR C Levitt, N Nouri, A E Jedlicka, et al.
Circulation|December 26, 2001
beta-adrenergic receptor blockers restore cardiac calcium release channel (ryanodine receptor) structure and function in heart failureS Reiken, M Gaburjakova, J Gaburjakova, et al.
Circulation|August 8, 2001
Comparison of right and left ventricular responses to left ventricular assist device support in patients with severe heart failure: a primary role of mechanical unloading underlying reverse remodelingA Barbone, J W Holmes, P M Heerdt, et al.
Translational Psychiatry|August 2, 2017
BACE1 inhibition by microdose lithium formulation NP03 rescues memory loss and early stage amyloid neuropathologyE N Wilson, S Do Carmo, M F Iulita, et al.
Circulation|November 30, 2000
Chronic unloading by left ventricular assist device reverses contractile dysfunction and alters gene expression in end-stage heart failureP M Heerdt, J W Holmes, B Cai, et al.
Biochemistry|August 10, 2000
Phosphatidylcholine activation of human heart (R)-3-hydroxybutyrate dehydrogenase mutants lacking active center sulfhydryls: site-directed mutagenesis of a new recombinant fusion proteinD Chelius, C Loeb-Hennard, S Fleischer, et al.
Pageof 8

Showing results (71-80 of 80) with videos related to

Sort By:
Pageof 8
You have reached the last page of results.This site can display upto 80 results.
Circulation Research|November 22, 2001
Dilated cardiomyopathy and sudden death resulting from constitutive activation of protein kinase aC L Antos, N Frey, S O Marx, et al.
Cell|May 20, 1994
Stabilization of calcium release channel (ryanodine receptor) function by FK506-binding proteinA B Brillantes, K Ondrias, A Scott, et al.
The Journal of Clinical Investigation|September 1, 1992
Expression of dihydropyridine receptor (Ca2+ channel) and calsequestrin genes in the myocardium of patients with end-stage heart failureT Takahashi, P D Allen, R V Lacro, et al.
The Journal of Biological Chemistry|June 3, 1994
Localization of the high and low affinity [3H]ryanodine binding sites on the skeletal muscle Ca2+ release channelC Callaway, A Seryshev, J P Wang, et al.
Genomics|November 1, 1991
Evidence for genetic heterogeneity in malignant hyperthermia susceptibilityR C Levitt, N Nouri, A E Jedlicka, et al.
Circulation|December 26, 2001
beta-adrenergic receptor blockers restore cardiac calcium release channel (ryanodine receptor) structure and function in heart failureS Reiken, M Gaburjakova, J Gaburjakova, et al.
Circulation|August 8, 2001
Comparison of right and left ventricular responses to left ventricular assist device support in patients with severe heart failure: a primary role of mechanical unloading underlying reverse remodelingA Barbone, J W Holmes, P M Heerdt, et al.
Translational Psychiatry|August 2, 2017
BACE1 inhibition by microdose lithium formulation NP03 rescues memory loss and early stage amyloid neuropathologyE N Wilson, S Do Carmo, M F Iulita, et al.
Circulation|November 30, 2000
Chronic unloading by left ventricular assist device reverses contractile dysfunction and alters gene expression in end-stage heart failureP M Heerdt, J W Holmes, B Cai, et al.
Biochemistry|August 10, 2000
Phosphatidylcholine activation of human heart (R)-3-hydroxybutyrate dehydrogenase mutants lacking active center sulfhydryls: site-directed mutagenesis of a new recombinant fusion proteinD Chelius, C Loeb-Hennard, S Fleischer, et al.
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