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The New England Journal of Medicine
|
November 18, 2000
The use of contrast-enhanced magnetic resonance imaging to identify reversible myocardial dysfunction
R J Kim, E Wu, A Rafael, et al.
Human Molecular Genetics
|
March 11, 1999
Characterization of dystrophin and utrophin diversity in the mouse
C N Lumeng, S F Phelps, J A Rafael, et al.
Journal of Equine Veterinary Science
|
August 8, 2025
Management of retained fetal membranes by umbilical vessel infusion in mares submitted to elective C-section
Marcos Eduardo Neto, Bruna R Curcio, Leandro A Rafael, et al.
Journal of Molecular and Cellular Cardiology
|
June 3, 2008
Claudin-5 levels are reduced in human end-stage cardiomyopathy
Tessily A Mays, Philip F Binkley, Amanda Lesinski, et al.
Brain : a Journal of Neurology
|
February 3, 2026
ARX mutation-associated interneuron defects provide insights into mechanisms underlying developmental epilepsies
Youngshin Lim, Shyam K Akula, Abigail K Myers, et al.
Frontiers in Physiology
|
January 24, 2022
Corrigendum: Muscle Twitch Kinetics Are Dependent on Muscle Group, Disease State, and Age in Duchenne Muscular Dystrophy Mouse Models
Kyra K Peczkowski, Neha Rastogi, Jeovanna Lowe, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology
|
July 17, 2015
Mineralocorticoid receptors are present in skeletal muscle and represent a potential therapeutic target
Jessica A Chadwick, J Spencer Hauck, Jeovanna Lowe, et al.
Frontiers in Physiology
|
October 26, 2020
Muscle Twitch Kinetics Are Dependent on Muscle Group, Disease State, and Age in Duchenne Muscular Dystrophy Mouse Models
Kyra K Peczkowski, Neha Rastogi, Jeovanna Lowe, et al.
Human Molecular Genetics
|
November 1, 2016
Myeloid cells are capable of synthesizing aldosterone to exacerbate damage in muscular dystrophy
Jessica A Chadwick, Sarah A Swager, Jeovanna Lowe, et al.
Human Molecular Genetics
|
May 23, 1998
mdx muscle pathology is independent of nNOS perturbation
R H Crosbie, V Straub, H Y Yun, et al.
Page
of 16
Search research articles
Search
Showing results (111-120 of 153) with videos related to
Sort By:
Page
of 16
The New England Journal of Medicine
|
November 18, 2000
The use of contrast-enhanced magnetic resonance imaging to identify reversible myocardial dysfunction
R J Kim, E Wu, A Rafael, et al.
Human Molecular Genetics
|
March 11, 1999
Characterization of dystrophin and utrophin diversity in the mouse
C N Lumeng, S F Phelps, J A Rafael, et al.
Journal of Equine Veterinary Science
|
August 8, 2025
Management of retained fetal membranes by umbilical vessel infusion in mares submitted to elective C-section
Marcos Eduardo Neto, Bruna R Curcio, Leandro A Rafael, et al.
Journal of Molecular and Cellular Cardiology
|
June 3, 2008
Claudin-5 levels are reduced in human end-stage cardiomyopathy
Tessily A Mays, Philip F Binkley, Amanda Lesinski, et al.
Brain : a Journal of Neurology
|
February 3, 2026
ARX mutation-associated interneuron defects provide insights into mechanisms underlying developmental epilepsies
Youngshin Lim, Shyam K Akula, Abigail K Myers, et al.
Frontiers in Physiology
|
January 24, 2022
Corrigendum: Muscle Twitch Kinetics Are Dependent on Muscle Group, Disease State, and Age in Duchenne Muscular Dystrophy Mouse Models
Kyra K Peczkowski, Neha Rastogi, Jeovanna Lowe, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology
|
July 17, 2015
Mineralocorticoid receptors are present in skeletal muscle and represent a potential therapeutic target
Jessica A Chadwick, J Spencer Hauck, Jeovanna Lowe, et al.
Frontiers in Physiology
|
October 26, 2020
Muscle Twitch Kinetics Are Dependent on Muscle Group, Disease State, and Age in Duchenne Muscular Dystrophy Mouse Models
Kyra K Peczkowski, Neha Rastogi, Jeovanna Lowe, et al.
Human Molecular Genetics
|
November 1, 2016
Myeloid cells are capable of synthesizing aldosterone to exacerbate damage in muscular dystrophy
Jessica A Chadwick, Sarah A Swager, Jeovanna Lowe, et al.
Human Molecular Genetics
|
May 23, 1998
mdx muscle pathology is independent of nNOS perturbation
R H Crosbie, V Straub, H Y Yun, et al.
Page
of 16