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Journal of Receptor Research|January 1, 1983
A method for dissecting two site receptor interactions in ligand binding studiesA Richardson, D R HowlettHistology and Histopathology|October 6, 2011
APP transgenic mice and their application to drug discoveryD R HowlettEuropean Journal of Pharmacology|April 29, 1986
Angiotensin-converting enzyme responses following enalapril in the sodium deficient ratS D Longman, D R HowlettBiochemical Pharmacology|November 1, 1988
Potassium efflux enhancement by cromakalim (BRL 34915) in rabbit mesenteric artery: an indirect effect independent of calcium?M C Coldwell, D R HowlettHistology and Histopathology|November 18, 2008
The pathology of APP transgenic mice: a model of Alzheimer's disease or simply overexpression of APP?D R Howlett, J C RichardsonArzneimittel-Forschung|May 1, 1988
Acute and chronic effects of a novel dihydrobenzofuran analogue and enalapril on blood pressure and plasma and tissue angiotensin converting enzyme activity in the sodium deficient normotensive ratS D Longman, D R HowlettBiopolymers|November 5, 1999
Computationally derived structural models of the beta-amyloid found in Alzheimer's disease plaques and the interaction with possible aggregation inhibitorsA R George, D R HowlettBiochemical Pharmacology|November 1, 1987
Specificity of action of the novel antihypertensive agent, BRL 34915, as a potassium channel activator. Comparison with nicorandilM C Coldwell, D R HowlettEuropean Journal of Pharmacology|December 7, 1979
Loss of beta-adrenoceptor binding sites in rat striatum following kainic acid lesionsS R Nahorski, D R Howlett, P RedgraveFEBS Letters|March 21, 1994
beta-Cyclodextrin interacts with the Alzheimer amyloid beta-A4 peptideP Camilleri, N J Haskins, D R HowlettPageof 227