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Showing results (451-460 of 455) with videos related to

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Cell|November 26, 2013
A polymorphism in IRF4 affects human pigmentation through a tyrosinase-dependent MITF/TFAP2A pathwayChristian Praetorius, Christine Grill, Simon N Stacey, et al.
The Journal of Clinical Investigation|May 4, 2021
The deacylase SIRT5 supports melanoma viability by influencing chromatin dynamicsWilliam Giblin, Lauren Bringman-Rodenbarger, Angela H Guo, et al.
Nature Communications|January 16, 2019
Publisher Correction: Novel pleiotropic risk loci for melanoma and nevus density implicate multiple biological pathwaysDavid L Duffy, Gu Zhu, Xin Li, et al.
Nature Communications|November 16, 2018
Novel pleiotropic risk loci for melanoma and nevus density implicate multiple biological pathwaysDavid L Duffy, Gu Zhu, Xin Li, et al.
Nature Genetics|April 29, 2020
Genome-wide association meta-analyses combining multiple risk phenotypes provide insights into the genetic architecture of cutaneous melanoma susceptibilityMaria Teresa Landi, D Timothy Bishop, Stuart MacGregor, et al.
Pageof 46

Showing results (451-460 of 455) with videos related to

Sort By:
Pageof 46
You have reached the last page of results.This site can display upto 455 results.
Cell|November 26, 2013
A polymorphism in IRF4 affects human pigmentation through a tyrosinase-dependent MITF/TFAP2A pathwayChristian Praetorius, Christine Grill, Simon N Stacey, et al.
The Journal of Clinical Investigation|May 4, 2021
The deacylase SIRT5 supports melanoma viability by influencing chromatin dynamicsWilliam Giblin, Lauren Bringman-Rodenbarger, Angela H Guo, et al.
Nature Communications|January 16, 2019
Publisher Correction: Novel pleiotropic risk loci for melanoma and nevus density implicate multiple biological pathwaysDavid L Duffy, Gu Zhu, Xin Li, et al.
Nature Communications|November 16, 2018
Novel pleiotropic risk loci for melanoma and nevus density implicate multiple biological pathwaysDavid L Duffy, Gu Zhu, Xin Li, et al.
Nature Genetics|April 29, 2020
Genome-wide association meta-analyses combining multiple risk phenotypes provide insights into the genetic architecture of cutaneous melanoma susceptibilityMaria Teresa Landi, D Timothy Bishop, Stuart MacGregor, et al.
Pageof 46