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A Samali

Showing results (31-40 of 39) with videos related to

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Cell Death & Disease|May 3, 2014
Nerve growth factor-mediated inhibition of apoptosis post-caspase activation is due to removal of active caspase-3 in a lysosome-dependent mannerK Mnich, L A Carleton, E T Kavanagh, et al.
Biochemical and Biophysical Research Communications|December 22, 1999
Phosphatidylserine exposure during apoptosis is a cell-type-specific event and does not correlate with plasma membrane phospholipid scramblase expressionB Fadeel, B Gleiss, K Högstrand, et al.
Cell Stress & Chaperones|August 30, 2001
Hsp27 protects mitochondria of thermotolerant cells against apoptotic stimuliA Samali, J D Robertson, E Peterson, et al.
Cell Death & Disease|June 26, 2015
RIPK1 promotes death receptor-independent caspase-8-mediated apoptosis under unresolved ER stress conditionsY Estornes, M A Aguileta, C Dubuisson, et al.
Cell Death & Disease|December 6, 2014
RIPK1 promotes death receptor-independent caspase-8-mediated apoptosis under unresolved ER stress conditionsY Estornes, M A Aguileta, C Dubuisson, et al.
Cell Death and Differentiation|June 19, 2012
PERK is required at the ER-mitochondrial contact sites to convey apoptosis after ROS-based ER stressT Verfaillie, N Rubio, A D Garg, et al.
Cell Death & Disease|June 29, 2012
Perk-dependent repression of miR-106b-25 cluster is required for ER stress-induced apoptosisS Gupta, D E Read, A Deepti, et al.
Cell Death & Disease|March 4, 2011
Rapid and efficient cancer cell killing mediated by high-affinity death receptor homotrimerizing TRAIL variantsC R Reis, A M van der Sloot, A Natoni, et al.
Oncogene|June 9, 2015
Decoy receptors block TRAIL sensitivity at a supracellular level: the role of stromal cells in controlling tumour TRAIL sensitivityL O'Leary, A M van der Sloot, C R Reis, et al.
Pageof 4

Showing results (31-40 of 39) with videos related to

Sort By:
Pageof 4
You have reached the last page of results.This site can display upto 39 results.
Cell Death & Disease|May 3, 2014
Nerve growth factor-mediated inhibition of apoptosis post-caspase activation is due to removal of active caspase-3 in a lysosome-dependent mannerK Mnich, L A Carleton, E T Kavanagh, et al.
Biochemical and Biophysical Research Communications|December 22, 1999
Phosphatidylserine exposure during apoptosis is a cell-type-specific event and does not correlate with plasma membrane phospholipid scramblase expressionB Fadeel, B Gleiss, K Högstrand, et al.
Cell Stress & Chaperones|August 30, 2001
Hsp27 protects mitochondria of thermotolerant cells against apoptotic stimuliA Samali, J D Robertson, E Peterson, et al.
Cell Death & Disease|June 26, 2015
RIPK1 promotes death receptor-independent caspase-8-mediated apoptosis under unresolved ER stress conditionsY Estornes, M A Aguileta, C Dubuisson, et al.
Cell Death & Disease|December 6, 2014
RIPK1 promotes death receptor-independent caspase-8-mediated apoptosis under unresolved ER stress conditionsY Estornes, M A Aguileta, C Dubuisson, et al.
Cell Death and Differentiation|June 19, 2012
PERK is required at the ER-mitochondrial contact sites to convey apoptosis after ROS-based ER stressT Verfaillie, N Rubio, A D Garg, et al.
Cell Death & Disease|June 29, 2012
Perk-dependent repression of miR-106b-25 cluster is required for ER stress-induced apoptosisS Gupta, D E Read, A Deepti, et al.
Cell Death & Disease|March 4, 2011
Rapid and efficient cancer cell killing mediated by high-affinity death receptor homotrimerizing TRAIL variantsC R Reis, A M van der Sloot, A Natoni, et al.
Oncogene|June 9, 2015
Decoy receptors block TRAIL sensitivity at a supracellular level: the role of stromal cells in controlling tumour TRAIL sensitivityL O'Leary, A M van der Sloot, C R Reis, et al.
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