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Journal of Neurophysiology|August 7, 2002
GDNF and NGF reverse changes in repriming of TTX-sensitive Na(+) currents following axotomy of dorsal root ganglion neuronsAndreas Leffler, Theodore R Cummins, Sulayman D Dib-Hajj, et al.
Neuron|April 1, 1993
Functional consequences of a Na+ channel mutation causing hyperkalemic periodic paralysisT R Cummins, J Zhou, F J Sigworth, et al.
Pflugers Archiv : European Journal of Physiology|December 22, 2016
FHF2 isoforms differentially regulate Nav1.6-mediated resurgent sodium currents in dorsal root ganglion neuronsCindy Barbosa, Yucheng Xiao, Andrew J Johnson, et al.
Neurobiology of Disease|January 18, 2012
Delayed calcium dysregulation in neurons requires both the NMDA receptor and the reverse Na+/Ca2+ exchangerMatthew K Brittain, Tatiana Brustovetsky, Patrick L Sheets, et al.
FEBS Letters|December 2, 1999
Two tetrodotoxin-resistant sodium channels in human dorsal root ganglion neuronsS D Dib-Hajj, L Tyrrell, T R Cummins, et al.
Proceedings of the National Academy of Sciences of the United States of America|May 17, 2006
A single sodium channel mutation produces hyper- or hypoexcitability in different types of neuronsAnthony M Rush, Sulayman D Dib-Hajj, Shujun Liu, et al.
Journal of Neurophysiology|April 4, 2002
HSV-1 helper virus 5dl1.2 suppresses sodium currents in amplicon-transduced neuronsBenjamin H White, Theodore R Cummins, Daniel H Wolf, et al.
Neuroscience Letters|April 2, 2016
A GABBR2 gene variant modifies pathophysiology in Huntington's diseaseApril L Philpott, Paul B Fitzgerald, Neil W Bailey, et al.
Journal of Clinical and Translational Science|February 22, 2024
Narrative review of telemedicine applications in decentralized researchMollie R Cummins, Hiral Soni, Julia Ivanova, et al.
Stem Cells (Dayton, Ohio)|March 22, 2016
Stepwise Differentiation of Retinal Ganglion Cells from Human Pluripotent Stem Cells Enables Analysis of Glaucomatous NeurodegenerationSarah K Ohlemacher, Akshayalakshmi Sridhar, Yucheng Xiao, et al.
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