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Revue Neurologique|January 1, 1991
Clinical syndromes associated with ragged red fibersL P Rowland, D M Blake, M Hirano, et al.
American Journal of Human Genetics|March 1, 1991
mtDNA depletion with variable tissue expression: a novel genetic abnormality in mitochondrial diseasesC T Moraes, S Shanske, H J Tritschler, et al.
Archives of Neurology|December 15, 2004
Studies of COX16, COX19, and PET191 in human cytochrome-c oxidase deficiencyStacey K H Tay, Claudia Nesti, Michelangelo Mancuso, et al.
Biorxiv : the Preprint Server for Biology|April 15, 2024
Premeiotic 24-nt phasiRNAs are present in the Zea genus and unique in biogenesis mechanism and molecular functionJunpeng Zhan, Sébastien Bélanger, Scott Lewis, et al.
Neurology|January 27, 1998
Multiple mtDNA deletions features in autosomal dominant and recessive diseases suggest distinct pathogenesesR Carrozzo, M Hirano, B Fromenty, et al.
Neurology|October 1, 1988
Immunocytochemical study of nebulin in Duchenne muscular dystrophyE Bonilla, A F Miranda, A Prelle, et al.
Nucleic Acids Research|February 11, 1990
Recombination via flanking direct repeats is a major cause of large-scale deletions of human mitochondrial DNAS Mita, R Rizzuto, C T Moraes, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|January 2, 2014
α-Synuclein is localized to mitochondria-associated ER membranesCristina Guardia-Laguarta, Estela Area-Gomez, Cornelia Rüb, et al.
Cell Death & Disease|June 10, 2024
Aberrant ER-mitochondria communication is a common pathomechanism in mitochondrial diseasePatricia Morcillo, Khushbu Kabra, Kevin Velasco, et al.
Proceedings of the National Academy of Sciences of the United States of America|May 13, 2024
Premeiotic 24-nt phasiRNAs are present in the Zea genus and unique in biogenesis mechanism and molecular functionJunpeng Zhan, Sébastien Bélanger, Scott Lewis, et al.
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