Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

A Shenker

Showing results (1-10 of 35) with videos related to

Pageof 4
Sort By:
Bailliere'S Clinical Endocrinology and Metabolism|July 1, 1995
G protein-coupled receptor structure and function: the impact of disease-causing mutationsA Shenker
Advances in Pediatrics|January 1, 1992
The mechanism of action of drugs used to treat attention-deficit hyperactivity disorder: focus on catecholamine receptor pharmacologyA Shenker
Clinical Biochemistry|October 1, 1993
G protein mutations in human diseaseL S Weinstein, A Shenker
The Journal of Biological Chemistry|December 13, 1996
The role of Asp578 in maintaining the inactive conformation of the human lutropin/choriogonadotropin receptorS Kosugi, T Mori, A Shenker
Protein Engineering|May 1, 1997
A model of the lutropin/choriogonadotropin receptor: insights into the structural and functional effects of constitutively activating mutationsZ Lin, A Shenker, R Pearlstein
Molecular Pharmacology|June 20, 1998
An anionic residue at position 564 is important for maintaining the inactive conformation of the human lutropin/choriogonadotropin receptorS Kosugi, T Mori, A Shenker
Pediatric Emergency Care|March 1, 1988
An unusual cause of right lower quadrant pain in a prepubertal childP Fosarelli, B Beaver, A Shenker
FEBS Letters|December 19, 1994
Constitutive activation of cyclic AMP but not phosphatidylinositol signaling caused by four mutations in the 6th transmembrane helix of the human thyrotropin receptorS Kosugi, A Shenker, T Mori
The Journal of Clinical Investigation|September 1, 1993
Abnormalities in G protein-coupled signal transduction pathways in human diseaseA M Spiegel, L S Weinstein, A Shenker
Endocrine Reviews|August 1, 1992
Receptor-effector coupling by G proteins: implications for normal and abnormal signal transductionA M Spiegel, A Shenker, L S Weinstein
Pageof 4

Showing results (1-10 of 35) with videos related to

Sort By:
Pageof 4
Bailliere'S Clinical Endocrinology and Metabolism|July 1, 1995
G protein-coupled receptor structure and function: the impact of disease-causing mutationsA Shenker
Advances in Pediatrics|January 1, 1992
The mechanism of action of drugs used to treat attention-deficit hyperactivity disorder: focus on catecholamine receptor pharmacologyA Shenker
Clinical Biochemistry|October 1, 1993
G protein mutations in human diseaseL S Weinstein, A Shenker
The Journal of Biological Chemistry|December 13, 1996
The role of Asp578 in maintaining the inactive conformation of the human lutropin/choriogonadotropin receptorS Kosugi, T Mori, A Shenker
Protein Engineering|May 1, 1997
A model of the lutropin/choriogonadotropin receptor: insights into the structural and functional effects of constitutively activating mutationsZ Lin, A Shenker, R Pearlstein
Molecular Pharmacology|June 20, 1998
An anionic residue at position 564 is important for maintaining the inactive conformation of the human lutropin/choriogonadotropin receptorS Kosugi, T Mori, A Shenker
Pediatric Emergency Care|March 1, 1988
An unusual cause of right lower quadrant pain in a prepubertal childP Fosarelli, B Beaver, A Shenker
FEBS Letters|December 19, 1994
Constitutive activation of cyclic AMP but not phosphatidylinositol signaling caused by four mutations in the 6th transmembrane helix of the human thyrotropin receptorS Kosugi, A Shenker, T Mori
The Journal of Clinical Investigation|September 1, 1993
Abnormalities in G protein-coupled signal transduction pathways in human diseaseA M Spiegel, L S Weinstein, A Shenker
Endocrine Reviews|August 1, 1992
Receptor-effector coupling by G proteins: implications for normal and abnormal signal transductionA M Spiegel, A Shenker, L S Weinstein
Pageof 4