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Journal of Medicinal Chemistry|August 13, 2019
Homotrimerization Approach in the Design of Thrombospondin-1 Mimetic Peptides with Improved Potency in Triggering Regulated Cell Death of Cancer CellsThomas Denèfle, Elodie Pramil, Luis Gómez-Morales, et al.
The Journal of Experimental Medicine|January 6, 2000
The HIV-1 viral protein R induces apoptosis via a direct effect on the mitochondrial permeability transition poreE Jacotot, L Ravagnan, M Loeffler, et al.
Leukemia|December 9, 2025
REL overexpression and sustained NF-κB signaling associated with 2p gain induce resistance to BTK inhibitors in Chronic Lymphocytic LeukemiaLéa Dehgane, Eliane Mallet, Constance Brasseur, et al.
Blood Advances|October 29, 2024
Epigenetic features support the diagnosis of B-cell prolymphocytic leukemia and identify 2 clinicobiological subtypesStella Charalampopoulou, Elise Chapiro, Ferran Nadeu, et al.
Molecular Metabolism|June 2, 2020
Mitochondrial AIF loss causes metabolic reprogramming, caspase-independent cell death blockade, embryonic lethality, and perinatal hydrocephalusLaure Delavallée, Navrita Mathiah, Lauriane Cabon, et al.
Cancer Medicine|May 9, 2019
Gain of the short arm of chromosome 2 (2p gain) has a significant role in drug-resistant chronic lymphocytic leukemiaFotini Kostopoulou, Clementine Gabillaud, Elise Chapiro, et al.
Nature|March 30, 2001
Essential role of the mitochondrial apoptosis-inducing factor in programmed cell deathN Joza, S A Susin, E Daugas, et al.
American Journal of Hematology|December 2, 2017
"Double-hit" chronic lymphocytic leukemia: An aggressive subgroup with 17p deletion and 8q24 gainElise Chapiro, Claude Lesty, Clémentine Gabillaud, et al.
Biomolecules|July 27, 2022
Keeping Cell Death Alive: An Introduction into the French Cell Death Research NetworkGabriel Ichim, Benjamin Gibert, Sahil Adriouch, et al.
British Journal of Haematology|December 14, 2020
Clinical and biological features of B-cell neoplasms with CDK6 translocations: an association with a subgroup of splenic marginal zone lymphomas displaying frequent CD5 expression, prolymphocytic cells, and TP53 abnormalitiesBaptiste Gailllard, Pascale Cornillet-Lefebvre, Quoc-Hung Le, et al.
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