Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

A Toh-E

Showing results (11-20 of 129) with videos related to

Pageof 13
Sort By:
Molecular & General Genetics : MGG|December 30, 1975
Genes coding for the structure of the acid phosphatases in Saccharomyces cerevisiaeA Toh-e, S Kakimoto
Biochimie|April 11, 2001
Genetic dissection of the yeast 26S proteasome: cell cycle defects caused by the Deltarpn9 mutationJ Takeuchi, A Toh-e
Molecular & General Genetics : MGG|September 30, 1999
Genetic evidence for interaction between components of the yeast 26S proteasome: combination of a mutation in RPN12 (a lid component gene) with mutations in RPT1 (an ATPase gene) causes synthetic lethalityJ Takeuchi, A Toh-e
Journal of Bacteriology|November 1, 1974
Characterization of a dominant, constitutive mutation, PHOO, for the repressible acid phosphatase synthesis in Saccharomyces cerevisiaeA Toh-E, Y Oshima
Current Genetics|February 1, 1992
A novel mutation occurring in the PHO80 gene suppresses the PHO4c mutations of Saccharomyces cerevisiaeH Okada, A Toh-e
Gene|May 1, 1992
Isolation and characterization of two novel ras superfamily genes in Saccharomyces cerevisiaeY Matsui, A Toh-e
Yeast (Chichester, England)|December 1, 1985
The PET18 locus of Saccharomyces cerevisiae: a complex locus containing multiple genesA Toh-e, Y Sahashi
Nucleic Acids Research|June 25, 1985
Physical and functional structure of a yeast plasmid, pSB3, isolated from Zygosaccharomyces bisporusA Toh-e, I Utatsu
Genes & Genetic Systems|April 2, 2002
Defects in glycosylphosphatidylinositol (GPI) anchor synthesis activate Hog1 kinase and confer copper-resistance in Saccharomyces cerevisisaeA Toh-e, T Oguchi
Molecular and Cellular Biology|December 1, 1992
Yeast RHO3 and RHO4 ras superfamily genes are necessary for bud growth, and their defect is suppressed by a high dose of bud formation genes CDC42 and BEM1Y Matsui, A Toh-E
Pageof 13

Showing results (11-20 of 129) with videos related to

Sort By:
Pageof 13
Molecular & General Genetics : MGG|December 30, 1975
Genes coding for the structure of the acid phosphatases in Saccharomyces cerevisiaeA Toh-e, S Kakimoto
Biochimie|April 11, 2001
Genetic dissection of the yeast 26S proteasome: cell cycle defects caused by the Deltarpn9 mutationJ Takeuchi, A Toh-e
Molecular & General Genetics : MGG|September 30, 1999
Genetic evidence for interaction between components of the yeast 26S proteasome: combination of a mutation in RPN12 (a lid component gene) with mutations in RPT1 (an ATPase gene) causes synthetic lethalityJ Takeuchi, A Toh-e
Journal of Bacteriology|November 1, 1974
Characterization of a dominant, constitutive mutation, PHOO, for the repressible acid phosphatase synthesis in Saccharomyces cerevisiaeA Toh-E, Y Oshima
Current Genetics|February 1, 1992
A novel mutation occurring in the PHO80 gene suppresses the PHO4c mutations of Saccharomyces cerevisiaeH Okada, A Toh-e
Gene|May 1, 1992
Isolation and characterization of two novel ras superfamily genes in Saccharomyces cerevisiaeY Matsui, A Toh-e
Yeast (Chichester, England)|December 1, 1985
The PET18 locus of Saccharomyces cerevisiae: a complex locus containing multiple genesA Toh-e, Y Sahashi
Nucleic Acids Research|June 25, 1985
Physical and functional structure of a yeast plasmid, pSB3, isolated from Zygosaccharomyces bisporusA Toh-e, I Utatsu
Genes & Genetic Systems|April 2, 2002
Defects in glycosylphosphatidylinositol (GPI) anchor synthesis activate Hog1 kinase and confer copper-resistance in Saccharomyces cerevisisaeA Toh-e, T Oguchi
Molecular and Cellular Biology|December 1, 1992
Yeast RHO3 and RHO4 ras superfamily genes are necessary for bud growth, and their defect is suppressed by a high dose of bud formation genes CDC42 and BEM1Y Matsui, A Toh-E
Pageof 13