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Genesis (New York, N.Y. : 2000)|June 30, 2005
Using a histone yellow fluorescent protein fusion for tagging and tracking endothelial cells in ES cells and miceStuart T Fraser, Anna-Katerina Hadjantonakis, Kenneth E Sahr, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|February 21, 2020
Impaired SMAD1/5 Mechanotransduction and Cx37 (Connexin37) Expression Enable Pathological Vessel Enlargement and ShuntingHanna M Peacock, Ashkan Tabibian, Nathan Criem, et al.
Cardiovascular Research|September 10, 2024
Smad1/5 is acetylated in the dorsal aortae of the mouse embryo before the onset of blood flow, driving early arterial gene expressionMargo Daems, Ljuba C Ponomarev, Rita Simoes-Faria, et al.
Journal of Biomechanics|June 21, 2024
Cell signaling and tissue remodeling in the pulmonary autograft after the Ross procedure: A computational studyLauranne Maes, Thibault Vervenne, Amber Hendrickx, et al.
Thrombosis Research|September 11, 2016
Absence of Pear1 does not affect murine platelet function in vivoMaarten Criel, Benedetta Izzi, Christophe Vandenbriele, et al.
Cardiovascular Diabetology|February 7, 2025
Mural cell dysfunction contributes to diastolic heart failure by promoting endothelial dysfunction and vessel remodellingMandy O J Grootaert, Alessandra Pasut, Jana Raman, et al.
Scientific Reports|September 25, 2020
The effect of different anaesthetics on echocardiographic evaluation of diastolic dysfunction in a heart failure with preserved ejection fraction modelIlona Cuijpers, Paolo Carai, Pedro Mendes-Ferreira, et al.
Circulation Research|June 13, 2020
B55α/PP2A Limits Endothelial Cell Apoptosis During Vascular Remodeling: A Complementary Approach To Disrupt Pathological Vessels?Manuel Ehling, Ward Celus, Rosa Martín-Pérez, et al.
European Heart Journal Open|January 4, 2024
Pericyte loss initiates microvascular dysfunction in the development of diastolic dysfunctionSteven J Simmonds, Mandy O J Grootaert, Ilona Cuijpers, et al.
Leukemia|November 7, 2008
Detection and molecular monitoring of FIP1L1-PDGFRA-positive disease by analysis of patient-specific genomic DNA fusion junctionsJ Score, C Walz, J V Jovanovic, et al.
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