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Eye (London, England)|February 28, 2009
Mortality in primary open-angle glaucoma: 'two cupped discs and a funeral'A W Hewitt, P Sanfilippo, M A Ring, et al.Journal of Medical Genetics|September 6, 2005
Lack of association of p53 polymorphisms and haplotypes in high and normal tension open angle glaucomaD P Dimasi, A W Hewitt, C M Green, et al.Current Opinion in Ophthalmology|October 28, 1999
Glaucoma genetics: where are we? Where will we go?J E Craig, D A MackeyThe British Journal of Ophthalmology|August 4, 2006
The role of the Met98Lys optineurin variant in inherited optic nerve diseasesJ E Craig, A W Hewitt, D P Dimasi, et al.The British Journal of Ophthalmology|July 26, 2008
The natural history of OPA1-related autosomal dominant optic atrophyA C Cohn, C Toomes, A W Hewitt, et al.Clinical Genetics|August 28, 2007
Prevalence of CYP1B1 mutations in Australian patients with primary congenital glaucomaD P Dimasi, A W Hewitt, T Straga, et al.American Journal of Ophthalmology|April 9, 2001
The Taa1 restriction enzyme provides a simple means to identify the Q368STOP mutation of the myocilin gene in primary open angle glaucomaP N Baird, J Dickinson, J E Craig, et al.Ophthalmic Genetics|March 23, 2001
Laboratory methods in ophthalmic genetics: obtaining DNA from patientsJ L Dickinson, M M Sale, J E Craig, et al.The British Journal of Ophthalmology|June 21, 2005
The telomere of human chromosome 1p contains at least two independent autosomal dominant congenital cataract genesJ D McKay, B Patterson, J E Craig, et al.Eye (London, England)|April 16, 2011
Distribution of conjunctival ultraviolet autofluorescence in a population-based study: the Norfolk Island Eye StudyJ C Sherwin, A W Hewitt, L S Kearns, et al.Pageof 11