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Journal of Hypertension|September 21, 2001
Endothelial nitric oxide synthase protein is reduced in the renal medulla of two-kidney, one-clip hypertensive ratsA Wickman, I J Andersson, J Jia, et al.Antimicrobial Agents and Chemotherapy|May 26, 2006
In vitro activities of DX-619 and comparison quinolones against gram-positive cocciPaul A Wickman, Jennifer A Black, Ellen Smith Moland, et al.Hypertension (Dallas, Tex. : 1979)|March 1, 1996
Cardiac insulin-like growth factor I and growth hormone receptor expression in renal hypertensionG Guron, P Friberg, A Wickman, et al.The Journal of Antimicrobial Chemotherapy|March 4, 2006
Levofloxacin/imipenem prevents the emergence of high-level resistance among Pseudomonas aeruginosa strains already lacking susceptibility to one or both drugsPhilip D Lister, Daniel J Wolter, Paul A Wickman, et al.Antimicrobial Agents and Chemotherapy|January 27, 2006
In vitro activity of DX-619, a novel des-fluoro(6) quinolone, against a panel of Streptococcus pneumoniae mutants with characterized resistance mechanismsPaul A Wickman, Ellen Smith Moland, Jennifer A Black, et al.Blood Pressure. Supplement|January 1, 1995
Cardiac hypertrophy and related growth processes: the role of insulin-like growth factor-IP Friberg, J Isgaard, H Wåhlander, et al.The Journal of Antimicrobial Chemotherapy|October 26, 2006
In vitro development of resistance to DX-619 and other quinolones in enterococciPaul A Wickman, Jennifer A Black, Ellen Smith Moland, et al.Hypertension (Dallas, Tex. : 1979)|January 1, 1997
Induction of growth hormone receptor and insulin-like growth factor-I mRNA in aorta and caval vein during hemodynamic challengeA Wickman, P Friberg, M A Adams, et al.Antimicrobial Agents and Chemotherapy|January 16, 2007
In vitro studies with DQ-113 and comparison fluoroquinolones to determine propensities to select resistance in gram-positive cocciSeong Geun Hong, Ellen Smith Moland, Paul A Wickman, et al.Kidney International|February 1, 1994
Renin-angiotensin system in neonatal rats: induction of a renal abnormality in response to ACE inhibition or angiotensin II antagonismP Friberg, B Sundelin, S O Bohman, et al.Pageof 3