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Journal of Immunology (Baltimore, Md. : 1950)|August 21, 2003
B220+ double-negative T cells suppress polyclonal T cell activation by a Fas-independent mechanism that involves inhibition of IL-2 productionAbdel Rahim A Hamad, Abdiaziz S Mohamood, Crystal J Trujillo, et al.
Proceedings of the National Academy of Sciences of the United States of America|April 11, 2023
A mutagenesis study of autoantigen optimization for potential T1D vaccine designYi Song, David R Bell, Rizwan Ahmed, et al.
Nephron. Clinical Practice|October 25, 2014
Intestinal microbiota-kidney cross talk in acute kidney injury and chronic kidney diseaseSanjeev Noel, Maria N Martina-Lingua, Samatha Bandapalle, et al.
International Immunology|June 14, 2006
Gld mutation of Fas ligand increases the frequency and up-regulates cell survival genes in CD25+CD4+ TR cellsAbdiaziz S Mohamood, Crystal J Trujillo, Dongfeng Zheng, et al.
Journal of the American Society of Nephrology : JASN|January 10, 2019
Activation and Proliferation of PD-1+ Kidney Double-Negative T Cells Is Dependent on Nonclassical MHC Proteins and IL-2Mohanraj Sadasivam, Sanjeev Noel, Sul A Lee, et al.
World Journal of Diabetes|January 16, 2020
Current understandings of the pathogenesis of type 1 diabetes: Genetics to environmentAdebola Matthew Giwa, Rizwan Ahmed, Zahra Omidian, et al.
Immunological Investigations|September 17, 2025
Myelin-Reactive TCR/IgM Dual-Expresser Lymphocytes in Multiple Sclerosis: Linking Pathogenesis to Anti-CD20 TherapyPrajita Paul, Marjan Behzadirad, Rafid Al Hallaf, et al.
European Journal of Immunology|August 25, 2015
Syndecan-1 identifies and controls the frequency of IL-17-producing naïve natural killer T (NKT17) cells in miceHong Dai, Ayesha Rahman, Ankit Saxena, et al.
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