Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Abram Katz

Showing results (31-40 of 72) with videos related to

Pageof 8
Sort By:
Acta Physiologica (Oxford, England)|January 5, 2022
Acute normobaric hypoxia blunts contraction-mediated mTORC1- and JNK-signaling in human skeletal muscleMarcus Moberg, William Apró, Oscar Horwath, et al.
Pflugers Archiv : European Journal of Physiology|April 16, 2004
Insulin-independent glycogen supercompensation in isolated mouse skeletal muscle: role of phosphorylase inactivationMarie E Sandström, Fabio Abbate, Daniel C Andersson, et al.
American Journal of Physiology. Cell Physiology|July 7, 2007
Activation of aconitase in mouse fast-twitch skeletal muscle during contraction-mediated oxidative stressShi-Jin Zhang, Marie E Sandström, Johanna T Lanner, et al.
The Journal of Physiology|September 10, 2003
Contraction-mediated glycogenolysis in mouse skeletal muscle lacking creatine kinase: the role of phosphorylase b activationAbram Katz, Daniel C Andersson, Josephine Yu, et al.
The Journal of Physiology|April 14, 2011
Mitochondrial production of reactive oxygen species contributes to the β-adrenergic stimulation of mouse cardiomycytesDaniel C Andersson, Jérémy Fauconnier, Takashi Yamada, et al.
Biochemical and Biophysical Research Communications|September 26, 2006
Respiratory chain dysfunction in skeletal muscle does not cause insulin resistanceAnna Wredenberg, Christoph Freyer, Marie E Sandström, et al.
Experimental Neurology|October 17, 2017
Exercise training attenuates experimental autoimmune encephalomyelitis by peripheral immunomodulation rather than direct neuroprotectionOfira Einstein, Nina Fainstein, Olga Touloumi, et al.
Cardiovascular Research|December 13, 2006
Insulin potentiates TRPC3-mediated cation currents in normal but not in insulin-resistant mouse cardiomyocytesJérémy Fauconnier, Johanna T Lanner, Ariane Sultan, et al.
Diabetes|July 28, 2005
Insulin and inositol 1,4,5-trisphosphate trigger abnormal cytosolic Ca2+ transients and reveal mitochondrial Ca2+ handling defects in cardiomyocytes of ob/ob miceJérémy Fauconnier, Johanna T Lanner, Shi-Jin Zhang, et al.
The EMBO Journal|October 13, 2007
Hypermetabolism in mice caused by the central action of an unliganded thyroid hormone receptor alpha1Maria Sjögren, Anneke Alkemade, Jens Mittag, et al.
Pageof 8

Showing results (31-40 of 72) with videos related to

Sort By:
Pageof 8
Acta Physiologica (Oxford, England)|January 5, 2022
Acute normobaric hypoxia blunts contraction-mediated mTORC1- and JNK-signaling in human skeletal muscleMarcus Moberg, William Apró, Oscar Horwath, et al.
Pflugers Archiv : European Journal of Physiology|April 16, 2004
Insulin-independent glycogen supercompensation in isolated mouse skeletal muscle: role of phosphorylase inactivationMarie E Sandström, Fabio Abbate, Daniel C Andersson, et al.
American Journal of Physiology. Cell Physiology|July 7, 2007
Activation of aconitase in mouse fast-twitch skeletal muscle during contraction-mediated oxidative stressShi-Jin Zhang, Marie E Sandström, Johanna T Lanner, et al.
The Journal of Physiology|September 10, 2003
Contraction-mediated glycogenolysis in mouse skeletal muscle lacking creatine kinase: the role of phosphorylase b activationAbram Katz, Daniel C Andersson, Josephine Yu, et al.
The Journal of Physiology|April 14, 2011
Mitochondrial production of reactive oxygen species contributes to the β-adrenergic stimulation of mouse cardiomycytesDaniel C Andersson, Jérémy Fauconnier, Takashi Yamada, et al.
Biochemical and Biophysical Research Communications|September 26, 2006
Respiratory chain dysfunction in skeletal muscle does not cause insulin resistanceAnna Wredenberg, Christoph Freyer, Marie E Sandström, et al.
Experimental Neurology|October 17, 2017
Exercise training attenuates experimental autoimmune encephalomyelitis by peripheral immunomodulation rather than direct neuroprotectionOfira Einstein, Nina Fainstein, Olga Touloumi, et al.
Cardiovascular Research|December 13, 2006
Insulin potentiates TRPC3-mediated cation currents in normal but not in insulin-resistant mouse cardiomyocytesJérémy Fauconnier, Johanna T Lanner, Ariane Sultan, et al.
Diabetes|July 28, 2005
Insulin and inositol 1,4,5-trisphosphate trigger abnormal cytosolic Ca2+ transients and reveal mitochondrial Ca2+ handling defects in cardiomyocytes of ob/ob miceJérémy Fauconnier, Johanna T Lanner, Shi-Jin Zhang, et al.
The EMBO Journal|October 13, 2007
Hypermetabolism in mice caused by the central action of an unliganded thyroid hormone receptor alpha1Maria Sjögren, Anneke Alkemade, Jens Mittag, et al.
Pageof 8