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Cells|May 28, 2022
Slack Potassium Channels Modulate TRPA1-Mediated Nociception in Sensory NeuronsFangyuan Zhou, Katharina Metzner, Patrick Engel, et al.
Journal of Neurochemistry|February 6, 2004
Protein associated with Myc (PAM) is involved in spinal nociceptive processingCorina Ehnert, Irmgard Tegeder, Sandra Pierre, et al.
Redox Report : Communications in Free Radical Research|August 25, 2025
Redox-dependent activation of protein kinase G1α contributes to transient receptor potential cation channel subfamily V member 1-mediated acute nociceptive pain behaviorTim Berg, Katharina Metzner, Nabil Bahrami, et al.
Antioxidants & Redox Signaling|January 24, 2014
Oxidant-induced activation of cGMP-dependent protein kinase Iα mediates neuropathic pain after peripheral nerve injuryJana E Lorenz, Wiebke Kallenborn-Gerhardt, Ruirui Lu, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 20, 2004
Reduced inflammatory hyperalgesia with preservation of acute thermal nociception in mice lacking cGMP-dependent protein kinase IIrmgard Tegeder, Domenico Del Turco, Achim Schmidtko, et al.
Neuropharmacology|August 22, 2017
K<sub>Ca</sub>3.1 channels modulate the processing of noxious chemical stimuli in miceRuirui Lu, Cathrin Flauaus, Lea Kennel, et al.
Molecular & Cellular Proteomics : MCP|August 30, 2008
Toponomics analysis of functional interactions of the ubiquitin ligase PAM (Protein Associated with Myc) during spinal nociceptive processingSandra Pierre, Christian Maeurer, Ovidiu Coste, et al.
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