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Nature Communications|July 2, 2014
Opposing effects of target overexpression reveal drug mechanismsAdam C Palmer, Roy KishonyNature Reviews. Genetics|February 20, 2013
Understanding, predicting and manipulating the genotypic evolution of antibiotic resistanceAdam C Palmer, Roy KishonyCell|December 16, 2017
Combination Cancer Therapy Can Confer Benefit via Patient-to-Patient Variability without Drug Additivity or SynergyAdam C Palmer, Peter K SorgerMolecular Cancer Therapeutics|December 17, 2025
Cross-resistance of Belinostat and Romidepsin in Non-T Follicular Helper Peripheral T-cell Lymphoma Models Suggests Subtype-Specific Implications for Belinostat-CHOPJacob C Pantazis, Adam C PalmerBlood Cancer Discovery|February 24, 2025
A Model of Intratumor and Interpatient Heterogeneity Explains Clinical Trials of Curative Combination Therapy for LymphomaAmy E Pomeroy, Adam C PalmerNature Chemical Biology|January 19, 2010
Chemical decay of an antibiotic inverts selection for resistanceAdam C Palmer, Elaine Angelino, Roy KishonyClinical Cancer Research : an Official Journal of the American Association for Cancer Research|December 18, 2023
Tumor-Specific Activity of Precision Medicines in the NCI-MATCH TrialIvvone Zhou, Deborah Plana, Adam C PalmerMolecular Biology and Evolution|September 1, 2018
Nonoptimal Gene Expression Creates Latent Potential for Antibiotic ResistanceAdam C Palmer, Remy Chait, Roy KishonyCell Systems|November 6, 2017
Regulatory Dynamics Determine Cell Fate following Abrupt Antibiotic ExposureDaniel Schultz, Adam C Palmer, Roy KishonyMedrxiv : the Preprint Server for Health Sciences|April 10, 2023
Tumor-specific activity of precision medicines in the NCI-MATCH trialIvvone Zhou, Deborah Plana, Adam C PalmerPageof 5