Showing results (1-10 of 10) with videos related to
Sort By:
Pageof 1
American Journal of Physiology. Endocrinology and Metabolism|May 14, 2009
Diabetes increases brain damage caused by severe hypoglycemiaAdam J Bree, Erwin C Puente, Dorit Daphna-Iken, et al.American Journal of Physiology. Endocrinology and Metabolism|May 7, 2009
Central but not systemic lipid infusion augments the counterregulatory response to hypoglycemiaSamuel C Haywood, Adam J Bree, Erwin C Puente, et al.Neuroscience Letters|January 29, 2011
Pharmacologic amelioration of severe hypoglycemia-induced neuronal damageJulie M Silverstein, Daniel Musikantow, Erwin C Puente, et al.Bone|August 30, 2011
Wnt6, Wnt10a and Wnt10b inhibit adipogenesis and stimulate osteoblastogenesis through a β-catenin-dependent mechanismWilliam P Cawthorn, Adam J Bree, Yao Yao, et al.Diabetes|January 21, 2010
Recurrent moderate hypoglycemia ameliorates brain damage and cognitive dysfunction induced by severe hypoglycemiaErwin C Puente, Julie Silverstein, Adam J Bree, et al.Diabetes|November 1, 2016
Brain GLUT4 Knockout Mice Have Impaired Glucose Tolerance, Decreased Insulin Sensitivity, and Impaired Hypoglycemic CounterregulationCandace M Reno, Erwin C Puente, Zhenyu Sheng, et al.Frontiers in Endocrinology|October 7, 2016
Increased Circulating Adiponectin in Response to Thiazolidinediones: Investigating the Role of Bone Marrow Adipose TissueRichard J Sulston, Brian S Learman, Bofeng Zhang, et al.Bone|January 24, 2018
Bone marrow adipocytes resist lipolysis and remodeling in response to β-adrenergic stimulationErica L Scheller, Shaima Khandaker, Brian S Learman, et al.Endocrinology|December 24, 2015
Expansion of Bone Marrow Adipose Tissue During Caloric Restriction Is Associated With Increased Circulating Glucocorticoids and Not With HypoleptinemiaWilliam P Cawthorn, Erica L Scheller, Sebastian D Parlee, et al.Cell Metabolism|July 8, 2014
Bone marrow adipose tissue is an endocrine organ that contributes to increased circulating adiponectin during caloric restrictionWilliam P Cawthorn, Erica L Scheller, Brian S Learman, et al.Pageof 1