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Adam R Wende

Showing results (51-60 of 106) with videos related to

Pageof 11
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Epigenetics|March 4, 2026
Transient elevation in cellular glucose uptake exacerbates pressure overload-induced cardiac hypertrophy and dysfunctionSayan Bakshi, Samuel F Chang, Luke A Potter, et al.
Plos One|July 27, 2010
Aberrant water homeostasis detected by stable isotope analysisShannon P O'Grady, Adam R Wende, Christopher H Remien, et al.
Scientific Reports|June 8, 2021
Identification of Nrf2-responsive microRNA networks as putative mediators of myocardial reductive stressJustin M Quiles, Mark E Pepin, Sini Sunny, et al.
Endocrinology|September 8, 2007
Cardiac hypertrophy caused by peroxisome proliferator- activated receptor-gamma agonist treatment occurs independently of changes in myocardial insulin signalingSandra Sena, Isaac R Rasmussen, Adam R Wende, et al.
Frontiers in Endocrinology|March 9, 2023
Vitamin A regulates tissue-specific organ remodeling in diet-induced obesity independent of mitochondrial functionIvanna Shymotiuk, Natali Froese, Christopher Werlein, et al.
Biological Psychiatry|August 6, 2019
The SETD6 Methyltransferase Plays an Essential Role in Hippocampus-Dependent Memory FormationWilliam M Webb, Ashleigh B Irwin, Mark E Pepin, et al.
Plos One|March 23, 2013
Metabolic determinants of electrical failure in ex-vivo canine model of cardiac arrest: evidence for the protective role of inorganic pyrophosphateJunko Shibayama, Tyson G Taylor, Paul W Venable, et al.
Endocrinology|May 27, 2010
Loss of bradykinin signaling does not accelerate the development of cardiac dysfunction in type 1 diabetic akita miceAdam R Wende, Jamie Soto, Curtis D Olsen, et al.
Journal of Molecular and Cellular Cardiology|June 7, 2011
Expression of slow skeletal TnI in adult mouse hearts confers metabolic protection to ischemiaKayla M Pound, Grace M Arteaga, Mathew Fasano, et al.
Frontiers in Cardiovascular Medicine|August 1, 2025
Severe attenuation of circadian clock output in the heart following sustained augmentation of cardiomyocyte protein O-GlcNAcylationGobinath Shanmugam, Samuel F Chang, Helen E Collins, et al.
Pageof 11

Showing results (51-60 of 106) with videos related to

Sort By:
Pageof 11
Epigenetics|March 4, 2026
Transient elevation in cellular glucose uptake exacerbates pressure overload-induced cardiac hypertrophy and dysfunctionSayan Bakshi, Samuel F Chang, Luke A Potter, et al.
Plos One|July 27, 2010
Aberrant water homeostasis detected by stable isotope analysisShannon P O'Grady, Adam R Wende, Christopher H Remien, et al.
Scientific Reports|June 8, 2021
Identification of Nrf2-responsive microRNA networks as putative mediators of myocardial reductive stressJustin M Quiles, Mark E Pepin, Sini Sunny, et al.
Endocrinology|September 8, 2007
Cardiac hypertrophy caused by peroxisome proliferator- activated receptor-gamma agonist treatment occurs independently of changes in myocardial insulin signalingSandra Sena, Isaac R Rasmussen, Adam R Wende, et al.
Frontiers in Endocrinology|March 9, 2023
Vitamin A regulates tissue-specific organ remodeling in diet-induced obesity independent of mitochondrial functionIvanna Shymotiuk, Natali Froese, Christopher Werlein, et al.
Biological Psychiatry|August 6, 2019
The SETD6 Methyltransferase Plays an Essential Role in Hippocampus-Dependent Memory FormationWilliam M Webb, Ashleigh B Irwin, Mark E Pepin, et al.
Plos One|March 23, 2013
Metabolic determinants of electrical failure in ex-vivo canine model of cardiac arrest: evidence for the protective role of inorganic pyrophosphateJunko Shibayama, Tyson G Taylor, Paul W Venable, et al.
Endocrinology|May 27, 2010
Loss of bradykinin signaling does not accelerate the development of cardiac dysfunction in type 1 diabetic akita miceAdam R Wende, Jamie Soto, Curtis D Olsen, et al.
Journal of Molecular and Cellular Cardiology|June 7, 2011
Expression of slow skeletal TnI in adult mouse hearts confers metabolic protection to ischemiaKayla M Pound, Grace M Arteaga, Mathew Fasano, et al.
Frontiers in Cardiovascular Medicine|August 1, 2025
Severe attenuation of circadian clock output in the heart following sustained augmentation of cardiomyocyte protein O-GlcNAcylationGobinath Shanmugam, Samuel F Chang, Helen E Collins, et al.
Pageof 11