Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Adrian Israelson

Showing results (21-30 of 40) with videos related to

Pageof 4
Sort By:
Journal of Neuroinflammation|January 11, 2024
CD4 T-cell aging exacerbates neuroinflammation in a late-onset mouse model of amyotrophic lateral sclerosisShir Zaccai, Anna Nemirovsky, Livnat Lerner, et al.
Scientific Reports|July 27, 2019
Cu/Zn-superoxide dismutase and wild-type like fALS SOD1 mutants produce cytotoxic quantities of H<sub>2</sub>O<sub>2</sub> via cysteine-dependent redox short-circuitShamchal Bakavayev, Nimrod Chetrit, Tatiana Zvagelsky, et al.
ACS Chemical Neuroscience|December 16, 2020
Exposure of β6/β7-Loop in Zn/Cu Superoxide Dismutase (SOD1) Is Coupled to Metal Loss and Is Transiently Reversible During MisfoldingShamchal Bakavayev, Shirel Argueti, Nachiyappan Venkatachalam, et al.
Proceedings of the National Academy of Sciences of the United States of America|August 24, 2016
Endogenous macrophage migration inhibitory factor reduces the accumulation and toxicity of misfolded SOD1 in a mouse model of ALSMarcel F Leyton-Jaimes, Clara Benaim, Salah Abu-Hamad, et al.
Neuron|August 28, 2010
Misfolded mutant SOD1 directly inhibits VDAC1 conductance in a mouse model of inherited ALSAdrian Israelson, Nir Arbel, Sandrine Da Cruz, et al.
Brain : a Journal of Neurology|July 3, 2023
Blocking an epitope of misfolded SOD1 ameliorates disease phenotype in a model of amyotrophic lateral sclerosisShamchal Bakavayev, Alexandra Stavsky, Shirel Argueti-Ostrovsky, et al.
Cell Death & Disease|January 27, 2018
MIF inhibits the formation and toxicity of misfolded SOD1 amyloid aggregates: implications for familial ALSNeta Shvil, Victor Banerjee, Guy Zoltsman, et al.
Journal of Cell Science|May 23, 2009
The VDAC1 N-terminus is essential both for apoptosis and the protective effect of anti-apoptotic proteinsSalah Abu-Hamad, Nir Arbel, Doron Calo, et al.
International Journal of Molecular Sciences|September 9, 2022
Targeting the Mitochondrial Protein VDAC1 as a Potential Therapeutic Strategy in ALSAnna Shteinfer-Kuzmine, Shirel Argueti-Ostrovsky, Marcel F Leyton-Jaimes, et al.
Biomolecules|February 25, 2023
Therapeutic Potential of Phytocannabinoid Cannabigerol for Multiple Sclerosis: Modulation of Microglial Activation In Vitro and In VivoSigal Fleisher-Berkovich, Yvonne Ventura, Maya Amoyal, et al.
Pageof 4

Showing results (21-30 of 40) with videos related to

Sort By:
Pageof 4
Journal of Neuroinflammation|January 11, 2024
CD4 T-cell aging exacerbates neuroinflammation in a late-onset mouse model of amyotrophic lateral sclerosisShir Zaccai, Anna Nemirovsky, Livnat Lerner, et al.
Scientific Reports|July 27, 2019
Cu/Zn-superoxide dismutase and wild-type like fALS SOD1 mutants produce cytotoxic quantities of H<sub>2</sub>O<sub>2</sub> via cysteine-dependent redox short-circuitShamchal Bakavayev, Nimrod Chetrit, Tatiana Zvagelsky, et al.
ACS Chemical Neuroscience|December 16, 2020
Exposure of β6/β7-Loop in Zn/Cu Superoxide Dismutase (SOD1) Is Coupled to Metal Loss and Is Transiently Reversible During MisfoldingShamchal Bakavayev, Shirel Argueti, Nachiyappan Venkatachalam, et al.
Proceedings of the National Academy of Sciences of the United States of America|August 24, 2016
Endogenous macrophage migration inhibitory factor reduces the accumulation and toxicity of misfolded SOD1 in a mouse model of ALSMarcel F Leyton-Jaimes, Clara Benaim, Salah Abu-Hamad, et al.
Neuron|August 28, 2010
Misfolded mutant SOD1 directly inhibits VDAC1 conductance in a mouse model of inherited ALSAdrian Israelson, Nir Arbel, Sandrine Da Cruz, et al.
Brain : a Journal of Neurology|July 3, 2023
Blocking an epitope of misfolded SOD1 ameliorates disease phenotype in a model of amyotrophic lateral sclerosisShamchal Bakavayev, Alexandra Stavsky, Shirel Argueti-Ostrovsky, et al.
Cell Death & Disease|January 27, 2018
MIF inhibits the formation and toxicity of misfolded SOD1 amyloid aggregates: implications for familial ALSNeta Shvil, Victor Banerjee, Guy Zoltsman, et al.
Journal of Cell Science|May 23, 2009
The VDAC1 N-terminus is essential both for apoptosis and the protective effect of anti-apoptotic proteinsSalah Abu-Hamad, Nir Arbel, Doron Calo, et al.
International Journal of Molecular Sciences|September 9, 2022
Targeting the Mitochondrial Protein VDAC1 as a Potential Therapeutic Strategy in ALSAnna Shteinfer-Kuzmine, Shirel Argueti-Ostrovsky, Marcel F Leyton-Jaimes, et al.
Biomolecules|February 25, 2023
Therapeutic Potential of Phytocannabinoid Cannabigerol for Multiple Sclerosis: Modulation of Microglial Activation In Vitro and In VivoSigal Fleisher-Berkovich, Yvonne Ventura, Maya Amoyal, et al.
Pageof 4