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Journal of Nuclear Medicine : Official Publication, Society of Nuclear Medicine|December 17, 2011
Preparation and characterization of L-[5-11C]-glutamine for metabolic imaging of tumorsWenchao Qu, Shunichi Oya, Brian P Lieberman, et al.
The Journal of Biological Chemistry|September 24, 2002
Bcl-x(L) complements Saccharomyces cerevisiae genes that facilitate the switch from glycolytic to oxidative metabolismMatthew G Vander Heiden, John S Choy, David J VanderWeele, et al.
Biochemical and Biophysical Research Communications|May 12, 2024
A high dose KRP203 induces cytoplasmic vacuoles associated with altered phosphoinositide segregation and endosome expansionSatoshi Kofuji, Kara Wolfe, Kazutaka Sumita, et al.
Cancer Research|March 18, 2004
Ovarian carcinoma expresses the NKG2D ligand Letal and promotes the survival and expansion of CD28- antitumor T cellsJose R Conejo-Garcia, Fabian Benencia, Maria C Courreges, et al.
Cancer Cell|November 17, 2012
ATF4 regulates MYC-mediated neuroblastoma cell death upon glutamine deprivationGuoliang Qing, Bo Li, Annette Vu, et al.
Virology|September 3, 2003
Bcl-2 and Bcl-xL overexpression inhibits cytochrome c release, activation of multiple caspases, and virus release following coxsackievirus B3 infectionChristopher M Carthy, Bobby Yanagawa, Honglin Luo, et al.
Proceedings of the National Academy of Sciences of the United States of America|November 23, 2011
Hypoxia promotes isocitrate dehydrogenase-dependent carboxylation of α-ketoglutarate to citrate to support cell growth and viabilityDavid R Wise, Patrick S Ward, Jessica E S Shay, et al.
Cancer Research|February 4, 2015
Human pancreatic cancer tumors are nutrient poor and tumor cells actively scavenge extracellular proteinJurre J Kamphorst, Michel Nofal, Cosimo Commisso, et al.
Cell Metabolism|July 28, 2015
Hypoxia Induces Production of L-2-HydroxyglutarateAndrew M Intlekofer, Raymond G Dematteo, Sriram Venneti, et al.
The Journal of Biological Chemistry|November 25, 2003
c-Myc sensitization to oxygen deprivation-induced cell death is dependent on Bax/Bak, but is independent of p53 and hypoxia-inducible factor-1Joslyn K Brunelle, Matthew T Santore, G R Scott Budinger, et al.
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