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Brain : a Journal of Neurology|July 24, 2021
Astrocyte-neuron interplay is critical for Alzheimer's disease pathogenesis and is rescued by TRPA1 channel blockadeAdrien Paumier, Sylvie Boisseau, Muriel Jacquier-Sarlin, et al.
Experimental Neurology|April 15, 2004
Neurotrophin-3-induced PI-3 kinase/Akt signaling rescues cortical neurons from apoptosisGéraldine Liot, Cécilia Gabriel, Mathias Cacquevel, et al.
Journal De La Societe De Biologie|August 13, 2003
[Does transforming growth factor-beta (TGF-beta) act as a neuroprotective agent in cerebral ischemia?]Fabian Docagne, Carine Ali, Sylvain Lesne, et al.
Organic & Biomolecular Chemistry|February 19, 2010
Copper-catalyzed amination of (bromophenyl)ethanolamine for a concise synthesis of aniline-containing analogues of NMDA NR2B antagonist ifenprodilCédric Bouteiller, Javier Becerril-Ortega, Patrice Marchand, et al.
Cellular and Molecular Neurobiology|September 30, 2003
Transforming growth factor-beta and ischemic brain injuryAlain Buisson, Sylvain Lesne, Fabian Docagne, et al.
The Journal of Biological Chemistry|March 31, 2005
Akt-dependent expression of NAIP-1 protects neurons against amyloid-{beta} toxicitySylvain Lesné, Cecilia Gabriel, Deirdre A Nelson, et al.
Journal of Alzheimer'S Disease : JAD|July 13, 2012
Selective impairment of some forms of synaptic plasticity by oligomeric amyloid-β peptide in the mouse hippocampus: implication of extrasynaptic NMDA receptorsMyriam Kervern, Arnaud Angeli, Olivier Nicole, et al.
FEBS Letters|May 14, 2008
P3 peptide, a truncated form of A beta devoid of synaptotoxic effect, does not assemble into soluble oligomersFabienne Dulin, Frédéric Léveillé, Javier Becerril Ortega, et al.
Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism|October 4, 2003
Reduction of ischemic brain damage by nitrous oxide and xenonHelene N David, Frederic Leveille, Laurent Chazalviel, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|April 25, 2014
Activity-dependent tau protein translocation to excitatory synapse is disrupted by exposure to amyloid-beta oligomersMarie Lise Frandemiche, Sandrine De Seranno, Travis Rush, et al.
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