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Human Molecular Genetics|January 9, 2009
Suppression of GFAP toxicity by alphaB-crystallin in mouse models of Alexander diseaseTracy L Hagemann, Wilbert C Boelens, Eric F Wawrousek, et al.The Journal of Biological Chemistry|February 23, 2017
Glial fibrillary acidic protein exhibits altered turnover kinetics in a mouse model of Alexander diseaseLaura R Moody, Gregory A Barrett-Wilt, Michael R Sussman, et al.Human Genetics|December 21, 2005
Propensity for paternal inheritance of de novo mutations in Alexander diseaseRong Li, Anne B Johnson, Gajja S Salomons, et al.Annals of the New York Academy of Sciences|November 1, 2017
P0-Cre Transgenic Mice for Inactivation of Adhesion Molecules in Schwann CellsMaria Laura Feltri, Maurizio D'Antonio, Stefano Previtali, et al.Journal of Neurochemistry|May 22, 2009
Dual transgenic reporter mice as a tool for monitoring expression of glial fibrillary acidic proteinWoosung Cho, Tracy L Hagemann, Delinda A Johnson, et al.Journal of Neurochemistry|November 5, 2004
Characterization of a Schwann cell enhancer in the myelin basic protein geneCarla Taveggia, Antonella Pizzagalli, Ernesta Fagiani, et al.Annals of the New York Academy of Sciences|November 1, 2017
Peripheral Nerve Dysmyelination Due to P0 Glycoprotein Overexpression Is Dose-DependentAngelo Quattrini, Maria Laura Feltri, Stefano Previtali, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|December 17, 2008
Nrf2 activation in astrocytes protects against neurodegeneration in mouse models of familial amyotrophic lateral sclerosisMarcelo R Vargas, Delinda A Johnson, Daniel W Sirkis, et al.ASN Neuro|October 10, 2013
Caspase cleavage of GFAP produces an assembly-compromised proteolytic fragment that promotes filament aggregationMei-Hsuan Chen, Tracy L Hagemann, Roy A Quinlan, et al.Nature Communications|November 27, 2015
Nitric oxide mediates glial-induced neurodegeneration in Alexander diseaseLiqun Wang, Tracy L Hagemann, Hermann Kalwa, et al.Pageof 17