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Nature Neuroscience|October 16, 2012
Lack of GPR88 enhances medium spiny neuron activity and alters motor- and cue-dependent behaviorsAlbert Quintana, Elisenda Sanz, Wengang Wang, et al.Biorxiv : the Preprint Server for Biology|November 24, 2025
Cell-Intrinsic Vulnerability and Immune Activation Cooperate to Drive Degeneration in a Mitochondrial Complex I Deficiency Model of Optic NeuropathyDaniela Santamaría-Muñoz, Raenier V Reyes, Miranda R Krueger, et al.Nature Communications|October 7, 2024
Compensatory activity of the PC-ME1 metabolic axis underlies differential sensitivity to mitochondrial complex I inhibitionLucia Del Prado, Myriam Jaraíz-Rodríguez, Mauro Agro, et al.Disease Models & Mechanisms|November 25, 2025
Generation and characterization of Col6a1 knock-in mice: A promising pre-clinical model for collagen VI-Related dystrophiesArístides López-Márquez, Carmen Badosa, Lluis Enjuanes-Ruiz, et al.Brain : a Journal of Neurology|December 1, 2021
Ndufs4 knockout mouse models of Leigh syndrome: pathophysiology and interventionMelissa A E van de Wal, Merel J W Adjobo-Hermans, Jaap Keijer, et al.Nature Communications|April 25, 2020
Functional and molecular heterogeneity of D2R neurons along dorsal ventral axis in the striatumEmma Puighermanal, Laia Castell, Anna Esteve-Codina, et al.Science (New York, N.Y.)|June 22, 2023
Splice variants of mitofusin 2 shape the endoplasmic reticulum and tether it to mitochondriaDéborah Naón, María Isabel Hernández-Alvarez, Satoko Shinjo, et al.Science (New York, N.Y.)|November 16, 2013
mTOR inhibition alleviates mitochondrial disease in a mouse model of Leigh syndromeSimon C Johnson, Melana E Yanos, Ernst-Bernhard Kayser, et al.Iscience|February 18, 2026
The succinate prodrug NV354 prevents brain lesions and late-stage motor dysfunction in mitochondrial complex I deficiencyMeagan J McManus, Yi Zhu, Cesar Alves, et al.Nature Communications|September 4, 2024
Cannabidiol ameliorates mitochondrial disease via PPARγ activation in preclinical modelsEmma Puighermanal, Marta Luna-Sánchez, Alejandro Gella, et al.Pageof 7