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Alberto Bosque

Showing results (71-80 of 88) with videos related to

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Retrovirology|August 22, 2014
Replication competent virus as an important source of bias in HIV latency models utilizing single round viral constructsPawel Bonczkowski, Ward De Spiegelaere, Alberto Bosque, et al.
Plos Pathogens|November 30, 2020
Human splice factors contribute to latent HIV infection in primary cell models and blood CD4+ T cells from ART-treated individualsSara Moron-Lopez, Sushama Telwatte, Indra Sarabia, et al.
Cell Reports|February 2, 2017
Benzotriazoles Reactivate Latent HIV-1 through Inactivation of STAT5 SUMOylationAlberto Bosque, Kyle A Nilson, Amanda B Macedo, et al.
Frontiers in Immunology|July 1, 2021
Activation of the Anti-Oxidative Stress Response Reactivates Latent HIV-1 Through the Mitochondrial Antiviral Signaling Protein Isoform MiniMAVSIndra Sarabia, Camille L Novis, Amanda B Macedo, et al.
Retrovirology|August 20, 2013
HIV latency and integration site placement in five cell-based modelsScott Sherrill-Mix, Mary K Lewinski, Marylinda Famiglietti, et al.
Biorxiv : the Preprint Server for Biology|April 27, 2026
Loss of Vpr-driven TRAIL-R2 expression protects HIV-infected cells from non-canonical NK cell TRAIL attackPaula E Grasberger, Abigail R Sondrini, Nicole Glidden, et al.
Iscience|October 20, 2023
AP-1/c-Fos supports SIV and HIV-1 latency in CD4 T cells infected <i>in vivo</i>Viviana Cobos Jiménez, Aviva Geretz, Andrey Tokarev, et al.
Journal of Virology|May 13, 2021
Selective BCL-X<sub>L</sub> Antagonists Eliminate Infected Cells from a Primary-Cell Model of HIV Latency but Not from <i>Ex Vivo</i> ReservoirsYanqin Ren, Szu Han Huang, Amanda B Macedo, et al.
Plos Pathogens|September 21, 2017
T-cell responses targeting HIV Nef uniquely correlate with infected cell frequencies after long-term antiretroviral therapyAllison S Thomas, Kimberley L Jones, Rajesh T Gandhi, et al.
Cell Host & Microbe|September 11, 2015
BIRC2/cIAP1 Is a Negative Regulator of HIV-1 Transcription and Can Be Targeted by Smac Mimetics to Promote Reversal of Viral LatencyLars Pache, Miriam S Dutra, Adam M Spivak, et al.
Pageof 9

Showing results (71-80 of 88) with videos related to

Sort By:
Pageof 9
Retrovirology|August 22, 2014
Replication competent virus as an important source of bias in HIV latency models utilizing single round viral constructsPawel Bonczkowski, Ward De Spiegelaere, Alberto Bosque, et al.
Plos Pathogens|November 30, 2020
Human splice factors contribute to latent HIV infection in primary cell models and blood CD4+ T cells from ART-treated individualsSara Moron-Lopez, Sushama Telwatte, Indra Sarabia, et al.
Cell Reports|February 2, 2017
Benzotriazoles Reactivate Latent HIV-1 through Inactivation of STAT5 SUMOylationAlberto Bosque, Kyle A Nilson, Amanda B Macedo, et al.
Frontiers in Immunology|July 1, 2021
Activation of the Anti-Oxidative Stress Response Reactivates Latent HIV-1 Through the Mitochondrial Antiviral Signaling Protein Isoform MiniMAVSIndra Sarabia, Camille L Novis, Amanda B Macedo, et al.
Retrovirology|August 20, 2013
HIV latency and integration site placement in five cell-based modelsScott Sherrill-Mix, Mary K Lewinski, Marylinda Famiglietti, et al.
Biorxiv : the Preprint Server for Biology|April 27, 2026
Loss of Vpr-driven TRAIL-R2 expression protects HIV-infected cells from non-canonical NK cell TRAIL attackPaula E Grasberger, Abigail R Sondrini, Nicole Glidden, et al.
Iscience|October 20, 2023
AP-1/c-Fos supports SIV and HIV-1 latency in CD4 T cells infected <i>in vivo</i>Viviana Cobos Jiménez, Aviva Geretz, Andrey Tokarev, et al.
Journal of Virology|May 13, 2021
Selective BCL-X<sub>L</sub> Antagonists Eliminate Infected Cells from a Primary-Cell Model of HIV Latency but Not from <i>Ex Vivo</i> ReservoirsYanqin Ren, Szu Han Huang, Amanda B Macedo, et al.
Plos Pathogens|September 21, 2017
T-cell responses targeting HIV Nef uniquely correlate with infected cell frequencies after long-term antiretroviral therapyAllison S Thomas, Kimberley L Jones, Rajesh T Gandhi, et al.
Cell Host & Microbe|September 11, 2015
BIRC2/cIAP1 Is a Negative Regulator of HIV-1 Transcription and Can Be Targeted by Smac Mimetics to Promote Reversal of Viral LatencyLars Pache, Miriam S Dutra, Adam M Spivak, et al.
Pageof 9