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Alberto Ferri

Showing results (21-30 of 81) with videos related to

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Journal of Neurochemistry|October 23, 2009
Inactivation of cytochrome c oxidase by mutant SOD1s in mouse motoneuronal NSC-34 cells is independent from copper availability but is because of nitric oxideMario Arciello, Concetta Rosa Capo, Mauro Cozzolino, et al.
Cells|July 2, 2021
Skeletal Muscle Metabolism: Origin or Prognostic Factor for Amyotrophic Lateral Sclerosis (ALS) Development?Cyril Quessada, Alexandra Bouscary, Frédérique René, et al.
Journal of Cachexia, Sarcopenia and Muscle|May 31, 2016
Improvement of skeletal muscle performance in ageing by the metabolic modulator TrimetazidineElisabetta Ferraro, Fabrizio Pin, Stefania Gorini, et al.
Plos One|June 6, 2013
CNF1 increases brain energy level, counteracts neuroinflammatory markers and rescues cognitive deficits in a murine model of Alzheimer's diseaseStefano Loizzo, Roberto Rimondini, Sara Travaglione, et al.
Plos One|October 13, 2015
CNF1 Enhances Brain Energy Content and Counteracts Spontaneous Epileptiform Phenomena in Aged DBA/2J MiceSara Travaglione, Giulia Ballan, Andrea Fortuna, et al.
Biometals : an International Journal on the Role of Metal Ions in Biology, Biochemistry, and Medicine|December 9, 2010
Copper depletion increases the mitochondrial-associated SOD1 in neuronal cellsMario Arciello, Concetta Rosa Capo, Sara D'Annibale, et al.
Antioxidants & Redox Signaling|April 7, 2009
Oligomerization of mutant SOD1 in mitochondria of motoneuronal cells drives mitochondrial damage and cell toxicityMauro Cozzolino, Maria Grazia Pesaresi, Ilaria Amori, et al.
International Journal of Molecular Sciences|September 23, 2022
Pattern of Mitochondrial Respiration in Peripheral Blood Cells of Patients with Parkinson's DiseaseTommaso Schirinzi, Illari Salvatori, Henri Zenuni, et al.
Cellular and Molecular Life Sciences : CMLS|July 31, 2023
An impaired splicing program underlies differentiation defects in hSOD1<sup>G93A</sup> neural progenitor cellsVeronica Verdile, Veronica Riccioni, Marika Guerra, et al.
Neurobiology of Disease|August 7, 2003
Mitochondrial dysfunction due to mutant copper/zinc superoxide dismutase associated with amyotrophic lateral sclerosis is reversed by N-acetylcysteineSimone Beretta, Gessica Sala, Laura Mattavelli, et al.
Pageof 9

Showing results (21-30 of 81) with videos related to

Sort By:
Pageof 9
Journal of Neurochemistry|October 23, 2009
Inactivation of cytochrome c oxidase by mutant SOD1s in mouse motoneuronal NSC-34 cells is independent from copper availability but is because of nitric oxideMario Arciello, Concetta Rosa Capo, Mauro Cozzolino, et al.
Cells|July 2, 2021
Skeletal Muscle Metabolism: Origin or Prognostic Factor for Amyotrophic Lateral Sclerosis (ALS) Development?Cyril Quessada, Alexandra Bouscary, Frédérique René, et al.
Journal of Cachexia, Sarcopenia and Muscle|May 31, 2016
Improvement of skeletal muscle performance in ageing by the metabolic modulator TrimetazidineElisabetta Ferraro, Fabrizio Pin, Stefania Gorini, et al.
Plos One|June 6, 2013
CNF1 increases brain energy level, counteracts neuroinflammatory markers and rescues cognitive deficits in a murine model of Alzheimer's diseaseStefano Loizzo, Roberto Rimondini, Sara Travaglione, et al.
Plos One|October 13, 2015
CNF1 Enhances Brain Energy Content and Counteracts Spontaneous Epileptiform Phenomena in Aged DBA/2J MiceSara Travaglione, Giulia Ballan, Andrea Fortuna, et al.
Biometals : an International Journal on the Role of Metal Ions in Biology, Biochemistry, and Medicine|December 9, 2010
Copper depletion increases the mitochondrial-associated SOD1 in neuronal cellsMario Arciello, Concetta Rosa Capo, Sara D'Annibale, et al.
Antioxidants & Redox Signaling|April 7, 2009
Oligomerization of mutant SOD1 in mitochondria of motoneuronal cells drives mitochondrial damage and cell toxicityMauro Cozzolino, Maria Grazia Pesaresi, Ilaria Amori, et al.
International Journal of Molecular Sciences|September 23, 2022
Pattern of Mitochondrial Respiration in Peripheral Blood Cells of Patients with Parkinson's DiseaseTommaso Schirinzi, Illari Salvatori, Henri Zenuni, et al.
Cellular and Molecular Life Sciences : CMLS|July 31, 2023
An impaired splicing program underlies differentiation defects in hSOD1<sup>G93A</sup> neural progenitor cellsVeronica Verdile, Veronica Riccioni, Marika Guerra, et al.
Neurobiology of Disease|August 7, 2003
Mitochondrial dysfunction due to mutant copper/zinc superoxide dismutase associated with amyotrophic lateral sclerosis is reversed by N-acetylcysteineSimone Beretta, Gessica Sala, Laura Mattavelli, et al.
Pageof 9