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Annals of Neurology|September 27, 2018
Prevalence of amyloid-β pathology in distinct variants of primary progressive aphasiaDavid Bergeron, Maria L Gorno-Tempini, Gil D Rabinovici, et al.Alzheimer'S Research & Therapy|March 28, 2024
A systematic review of progranulin concentrations in biofluids in over 7,000 people-assessing the pathogenicity of GRN mutations and other influencing factorsImogen J Swift, Rosa Rademakers, NiCole Finch, et al.Journal of Alzheimer'S Disease : JAD|November 14, 2025
Associations of lifestyle factors with amyloid pathology in persons without dementiaJulie E Oomens, Stephanie Jb Vos, Nancy N Maserejian, et al.Nature Genetics|November 21, 2022
Exome sequencing identifies rare damaging variants in ATP8B4 and ABCA1 as risk factors for Alzheimer's diseaseHenne Holstege, Marc Hulsman, Camille Charbonnier, et al.Neurology|September 18, 2020
C9orf72, age at onset, and ancestry help discriminate behavioral from language variants in FTLD cohortsBeatrice Costa, Claudia Manzoni, Manuel Bernal-Quiros, et al.Acta Neuropathologica|May 28, 2019
A nonsynonymous mutation in PLCG2 reduces the risk of Alzheimer's disease, dementia with Lewy bodies and frontotemporal dementia, and increases the likelihood of longevitySven J van der Lee, Olivia J Conway, Iris Jansen, et al.Acta Neuropathologica|January 20, 2020
Correction to: A nonsynonymous mutation in PLCG2 reduces the risk of Alzheimer's disease, dementia with Lewy bodies and frontotemporal dementia, and increases the likelihood of longevitySven J van der Lee, Olivia J Conway, Iris Jansen, et al.JAMA|July 13, 2026
Amyloid PET Quantitation and Centiloid Thresholds in the Diagnosis of Alzheimer Disease: An Individual Participant Data Meta-AnalysisGanna Blazhenets, David N Soleimani-Meigooni, Konstantinos Chiotis, et al.JAMA Psychiatry|December 1, 2017
Association of Cerebral Amyloid-β Aggregation With Cognitive Functioning in Persons Without DementiaWillemijn J Jansen, Rik Ossenkoppele, Betty M Tijms, et al.JAMA Psychiatry|January 22, 2025
Depressive Symptoms and Amyloid PathologyWietse A Wiels, Julie E Oomens, Sebastiaan Engelborghs, et al.Pageof 35