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Viruses|March 30, 2023
EBV Reactivation from Latency Is a Degrading Experience for the HostAlejandro Casco, Eric JohannsenCell Reports|September 19, 2024
Epstein-Barr virus induces host shutoff extensively via BGLF5-independent mechanismsAlejandro Casco, Makoto Ohashi, Eric JohannsenJournal of Virology|October 27, 2021
Accurate Quantification of Overlapping Herpesvirus Transcripts from RNA Sequencing DataAlejandro Casco, Akansha Gupta, Mitchell Hayes, et al.Plos Pathogens|September 29, 2022
Rta is the principal activator of Epstein-Barr virus epithelial lytic transcriptionAhmed Ali, Makoto Ohashi, Alejandro Casco, et al.Plos Pathogens|June 5, 2018
CAGE-seq analysis of Epstein-Barr virus lytic gene transcription: 3 kinetic classes from 2 mechanismsReza Djavadian, Mitchell Hayes, Eric JohannsenPlos Pathogens|June 28, 2016
An Epstein-Barr Virus-Encoded Protein Complex Requires an Origin of Lytic Replication In Cis to Mediate Late Gene TranscriptionReza Djavadian, Ya-Fang Chiu, Eric JohannsenJournal of Virology|June 20, 2008
The Epstein-Barr virus LF2 protein inhibits viral replicationMichael A Calderwood, Amy M Holthaus, Eric JohannsenPlos Pathogens|March 15, 2021
Epstein-Barr virus nuclear antigen 3C (EBNA3C) interacts with the metabolism sensing C-terminal binding protein (CtBP) repressor to upregulate host genesMakoto Ohashi, Mitchell Hayes, Kyle McChesney, et al.Journal of Virology|December 12, 2001
Epstein-Barr virus nuclear antigen 3C putative repression domain mediates coactivation of the LMP1 promoter with EBNA-2Jeffrey Lin, Eric Johannsen, Erle Robertson, et al.Pageof 4