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Cells|February 25, 2022
RNAi Screening Uncovers a Synthetic Sick Interaction between CtIP and the BARD1 Tumor SuppressorHella A Bolck, Sara Przetocka, Roger Meier, et al.Scientific Reports|November 10, 2017
Impaired oxidative stress response characterizes HUWE1-promoted X-linked intellectual disabilityMatthias Bosshard, Rossana Aprigliano, Cristina Gattiker, et al.Molecular Cell|January 15, 2021
Mitochondrial NAD+ Controls Nuclear ARTD1-Induced ADP-RibosylationAnn-Katrin Hopp, Federico Teloni, Lavinia Bisceglie, et al.Nature Communications|November 14, 2020
CHD7 and 53BP1 regulate distinct pathways for the re-ligation of DNA double-strand breaksMagdalena B Rother, Stefania Pellegrino, Rebecca Smith, et al.Nucleic Acids Research|November 9, 2022
PARP1 proximity proteomics reveals interaction partners at stressed replication forksThorsten Mosler, H Irem Baymaz, Justus F Gräf, et al.Science Advances|July 31, 2021
FAN1-MLH1 interaction affects repair of DNA interstrand cross-links and slipped-CAG/CTG repeatsAntonio Porro, Mohiuddin Mohiuddin, Christina Zurfluh, et al.Biorxiv : the Preprint Server for Biology|September 21, 2023
The Fanconi anemia core complex promotes CtIP-dependent end-resection to drive homologous recombination at DNA double-strand breaksBert van de Kooij, Fenna J van der Wal, Magdalena B Rother, et al.Nature Cell Biology|July 28, 2014
The NBS1-Treacle complex controls ribosomal RNA transcription in response to DNA damageDorthe H Larsen, Flurina Hari, Julie A Clapperton, et al.Nature Communications|August 16, 2024
The Fanconi anemia core complex promotes CtIP-dependent end resection to drive homologous recombination at DNA double-strand breaksBert van de Kooij, Fenna J van der Wal, Magdalena B Rother, et al.Cell|August 14, 2012
TRIP12 and UBR5 suppress spreading of chromatin ubiquitylation at damaged chromosomesThorkell Gudjonsson, Matthias Altmeyer, Velibor Savic, et al.Pageof 8