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Life (Basel, Switzerland)|June 2, 2021
Exploring the Release of Toxic Oligomers from α-Synuclein Fibrils with Antibodies and STED MicroscopyAlessandra Bigi, Emilio Ermini, Serene W Chen, et al.
Cellular & Molecular Biology Letters|July 12, 2024
Biophysical characterization of the phase separation of TDP-43 devoid of the C-terminal domainTommaso Staderini, Alessandra Bigi, Clément Lagrève, et al.
International Journal of Molecular Sciences|November 10, 2019
Identification of Novel 1,3,5-Triphenylbenzene Derivative Compounds as Inhibitors of Hen Lysozyme Amyloid Fibril FormationHassan Ramshini, Reza Tayebee, Alessandra Bigi, et al.
Glycobiology|September 14, 2013
A proline-rich loop mediates specific functions of human sialidase NEU4 in SK-N-BE neuronal differentiationAlessandra Bigi, Cristina Tringali, Matilde Forcella, et al.
Journal of Medicinal Chemistry|February 15, 2012
Synthesis and biological evaluation of 1,4-diaryl-2-azetidinones as specific anticancer agents: activation of adenosine monophosphate activated protein kinase and induction of apoptosisFarida Tripodi, Roberto Pagliarin, Gabriele Fumagalli, et al.
Glycobiology|October 3, 2009
Human sialidase NEU4 long and short are extrinsic proteins bound to outer mitochondrial membrane and the endoplasmic reticulum, respectivelyAlessandra Bigi, Lavinia Morosi, Chiara Pozzi, et al.
Journal of Alzheimer'S Disease : JAD|September 19, 2017
Soluble Oligomers Require a Ganglioside to Trigger Neuronal Calcium OverloadRoberta Cascella, Elisa Evangelisti, Alessandra Bigi, et al.
Mechanisms of Ageing and Development|September 26, 2025
Human astrocytes from healthy individuals and Alzheimer's patients respond differently to Aβ<sub>1-42</sub> oligomers, triggering distinct paths of reactivity and senescenceSara Ristori, Gianmarco Bertoni, Alessandra Bigi, et al.
Alzheimer'S Research & Therapy|January 18, 2024
A single-domain antibody detects and neutralises toxic Aβ<sub>42</sub> oligomers in the Alzheimer's disease CSFAlessandra Bigi, Liliana Napolitano, Devkee M Vadukul, et al.
The FEBS Journal|July 19, 2022
Sphingosine 1-phosphate attenuates neuronal dysfunction induced by amyloid-β oligomers through endocytic internalization of NMDA receptorsAlessandra Bigi, Roberta Cascella, Giulia Fani, et al.
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