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Cardiovascular Research|May 22, 2024
Gene therapy with phosphodiesterases 2A and 4B ameliorates heart failure and arrhythmias by improving subcellular cAMP compartmentationNikoleta Pavlaki, Alexander Froese, Wener Li, et al.
Science Signaling|November 20, 2014
Combined inhibition of PI3Kβ and PI3Kγ reduces fat mass by enhancing α-MSH-dependent sympathetic driveAlessia Perino, Martina Beretta, Ana Kilić, et al.
Science Signaling|December 22, 2016
Rac signal adaptation controls neutrophil mobilization from the bone marrowCarlo Cosimo Campa, Giulia Germena, Elisa Ciraolo, et al.
Scientific Reports|May 10, 2018
Hyperactivity of Rac1-GTPase pathway impairs neuritogenesis of cortical neurons by altering actin dynamicsValentina Zamboni, Maria Armentano, Gaia Berto, et al.
Scientific Reports|October 8, 2016
Disruption of ArhGAP15 results in hyperactive Rac1, affects the architecture and function of hippocampal inhibitory neurons and causes cognitive deficitsValentina Zamboni, Maria Armentano, Gabriella Sarò, et al.
Pharmacological Research|March 30, 2021
Understanding the heart-brain axis response in COVID-19 patients: A suggestive perspective for therapeutic developmentVincenzo Lionetti, Sveva Bollini, Raffaele Coppini, et al.
Hemasphere|March 6, 2023
In Humanized Sickle Cell Mice, Imatinib Protects Against Sickle Cell-Related InjuryEnrica Federti, Alessandro Matte, Antonio Recchiuti, et al.
Circulation|September 26, 2012
Phosphoinositide 3-kinase γ protects against catecholamine-induced ventricular arrhythmia through protein kinase A-mediated regulation of distinct phosphodiesterasesAlessandra Ghigo, Alessia Perino, Hind Mehel, et al.
European Journal of Heart Failure|March 8, 2019
The novel butyrate derivative phenylalanine-butyramide protects from doxorubicin-induced cardiotoxicityMichele Russo, Fiorentina Guida, Lorella Paparo, et al.
Cellular and Molecular Life Sciences : CMLS|August 11, 2023
MST1 mediates doxorubicin-induced cardiomyopathy by SIRT3 downregulationLeonardo Schirone, Daniele Vecchio, Valentina Valenti, et al.
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