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Frontiers in Endocrinology|July 26, 2021
Exposure to Perfluoroalkyl Chemicals and Cardiovascular Disease: Experimental and Epidemiological EvidenceAlessandra Meneguzzi, Cristiano Fava, Marco Castelli, et al.Frontiers in Pharmacology|November 30, 2019
Platelet-Derived Extracellular Vesicles as Target of Antiplatelet Agents. What Is the Evidence?Francesco Taus, Alessandra Meneguzzi, Marco Castelli, et al.Frontiers in Pharmacology|October 4, 2016
Antiplatelet Agents Inhibit the Generation of Platelet-Derived MicroparticlesAlice Giacomazzi, Maurizio Degan, Stefano Calabria, et al.Liver International : Official Journal of the International Association for the Study of the Liver|July 28, 2025
Platelet Functional Profile Is Altered in Metabolic Dysfunction-Associated Steatotic Liver DiseaseMarco Castelli, Mirko Zoncapè, Alessandra Meneguzzi, et al.Circulation|December 11, 2002
NCX-4016 (NO-aspirin) inhibits lipopolysaccharide-induced tissue factor expression in vivo: role of nitric oxideStefano Fiorucci, Andrea Mencarelli, Alessandra Meneguzzi, et al.Journal of the American College of Cardiology|September 11, 2004
Co-administration of nitric oxide-aspirin (NCX-4016) and aspirin prevents platelet and monocyte activation and protects against gastric damage induced by aspirin in humansStefano Fiorucci, Andrea Mencarelli, Alessandra Meneguzzi, et al.Journal of Hypertension|July 20, 2002
Collagen I and III mRNA gene expression and cell growth potential of skin fibroblasts in patients with essential hypertensionPietro Delva, Alessandro Lechi, Caterina Pastori, et al.Frontiers in Pharmacology|October 16, 2018
Calcium-Dependent Src Phosphorylation and Reactive Oxygen Species Generation Are Implicated in the Activation of Human Platelet Induced by Thromboxane A2 AnalogsPietro Minuz, Alessandra Meneguzzi, Laura Fumagalli, et al.Environment International|April 25, 2021
Interference of C6O4 on platelet aggregation pathways: Cues on the new-generation of perfluoro-alkyl substancePietro Minuz, Luca De Toni, Stefano Dall'Acqua, et al.Free Radical Biology & Medicine|April 27, 2013
Overexpression of TNF-α in mitochondrial diseases caused by mutations in mtDNA: evidence for signaling through its receptors on mitochondriaGaetano Vattemi, Matteo Marini, Nicholas R Ferreri, et al.Pageof 2