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ACS Nano|December 11, 2025
Multivalent Interactions between Chaperone and Ribosome-Nascent Chain Complex Revealed by High-Speed AFM and MD SimulationsEider Nuñez, Prithwidip Saha, Markel G Ibarluzea, et al.
Epilepsia|November 28, 2018
Homomeric Kv7.2 current suppression is a common feature in KCNQ2 epileptic encephalopathyCarolina Gomis-Pérez, Janire Urrutia, Anna Marcé-Grau, et al.
Elife|February 21, 2023
Redox regulation of KV7 channels through EF3 hand of calmodulinEider Nuñez, Frederick Jones, Arantza Muguruza-Montero, et al.
Protein Science : a Publication of the Protein Society|August 15, 2021
Do calmodulin binding IQ motifs have built-in capping domains?Arantza Muguruza-Montero, Rafael Ramis, Eider Nuñez, et al.
Protein Science : a Publication of the Protein Society|April 8, 2026
Calmodulin assists during co-translational folding of the KV7.2 channel calcium responsive domainArantza Muguruza-Montero, Jack R Tait, Sara M-Alicante, et al.
Human Genetics|November 22, 2007
A novel KCNQ4 pore-region mutation (p.G296S) causes deafness by impairing cell-surface channel expressionAngeles Mencía, Daniel González-Nieto, Silvia Modamio-Høybjør, et al.
BMC Biology|May 22, 2021
An epilepsy-causing mutation leads to co-translational misfolding of the Kv7.2 channelJanire Urrutia, Alejandra Aguado, Carolina Gomis-Perez, et al.
Scientific Reports|December 2, 2016
Early-onset epileptic encephalopathy caused by a reduced sensitivity of Kv7.2 potassium channels to phosphatidylinositol 4,5-bisphosphateMaria Virginia Soldovieri, Paolo Ambrosino, Ilaria Mosca, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|May 24, 2014
Functional assembly of Kv7.1/Kv7.5 channels with emerging properties on vascular muscle physiologyAnna Oliveras, Meritxell Roura-Ferrer, Laura Solé, et al.
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