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The Journal of Clinical Investigation|October 3, 2003
Inhibition of GAPDH activity by poly(ADP-ribose) polymerase activates three major pathways of hyperglycemic damage in endothelial cellsXueliang Du, Takeshi Matsumura, Diane Edelstein, et al.
American Journal of Physiology. Cell Physiology|March 18, 2024
Kynurenine 3-monooxygenase limits de novo NAD+ synthesis through dietary tryptophan in renal proximal tubule epithelial cell modelsYougang Zhai, Jose A Chavez, Katharine E D'Aquino, et al.
Atherosclerosis|January 5, 2002
Hyperglycemia induces PAI-1 gene expression in adipose tissue by activation of the hexosamine biosynthetic pathwayIlan Gabriely, Xiao Man Yang, Jane A Cases, et al.
The Journal of Experimental Medicine|September 24, 2008
Transient high glucose causes persistent epigenetic changes and altered gene expression during subsequent normoglycemiaAssam El-Osta, Daniella Brasacchio, Dachun Yao, et al.
Frontiers in Endocrinology|December 9, 2022
A FFAR1 full agonist restores islet function in models of impaired glucose-stimulated insulin secretion and diabetic non-human primatesBrian Rady, Jianying Liu, Hui Huang, et al.
Drug Discovery Today|January 19, 2016
Translational Medicine Guide transforms drug development processes: the recent Merck experienceHugues Dolgos, Mark Trusheim, Dietmar Gross, et al.
Molecular and Cellular Neurosciences|August 11, 2007
Identification and characterization of a novel phosphorylation site on the GluR1 subunit of AMPA receptorsHey-Kyoung Lee, Kogo Takamiya, Kimihiko Kameyama, et al.
Diabetes|June 11, 2004
Abnormal glucose homeostasis due to chronic hyperresistinemiaShamina M Rangwala, A Sophie Rich, Ben Rhoades, et al.
The Journal of Clinical Investigation|May 3, 2005
Severe impairment in liver insulin signaling fails to alter hepatic insulin action in conscious miceChristoph Buettner, Rima Patel, Evan D Muse, et al.
Cell Metabolism|October 9, 2008
SirT1 gain of function increases energy efficiency and prevents diabetes in miceAlexander S Banks, Ning Kon, Colette Knight, et al.
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