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Experimental Diabetes Research|November 24, 2011
Mitochondrial dysfunction and β-cell failure in type 2 diabetes mellitusZhongmin Alex Ma, Zhengshan Zhao, John TurkJournal of Cell Science|November 23, 2007
The increase of cell-membranous phosphatidylcholines containing polyunsaturated fatty acid residues induces phosphorylation of p53 through activation of ATRXu Hannah Zhang, Chunying Zhao, Zhongmin Alex MaThe Journal of Biological Chemistry|December 24, 2011
FTY720 normalizes hyperglycemia by stimulating β-cell in vivo regeneration in db/db mice through regulation of cyclin D3 and p57(KIP2)Zhengshan Zhao, Jinwoo Choi, Chunying Zhao, et al.Molecular Endocrinology (Baltimore, Md.)|October 9, 2004
Inhibition of Ca2+-independent phospholipase A2 results in insufficient insulin secretion and impaired glucose toleranceKeying Song, Xu Zhang, Chunying Zhao, et al.Plos Computational Biology|May 18, 2026
The speed limit of visual perception: Bidirectional influence of image memorability and processing speed on perceived duration and recognitionMartin Wiener, Chloe Mondok, Alex Ma, et al.The Journal of Biological Chemistry|August 24, 2004
An essential role of the Jak-2/STAT-3/cytosolic phospholipase A(2) axis in platelet-derived growth factor BB-induced vascular smooth muscle cell motilityIndira Neeli, Zhimin Liu, Nagadhara Dronadula, et al.The Journal of Biological Chemistry|May 27, 2006
Calcium-independent phospholipase A2 localizes in and protects mitochondria during apoptotic induction by staurosporineKonstantin Seleznev, Chunying Zhao, Xu Hannah Zhang, et al.Plos One|November 3, 2011
Genetic ablation of PLA2G6 in mice leads to cerebellar atrophy characterized by Purkinje cell loss and glial cell activationZhengshan Zhao, Jing Wang, Chunying Zhao, et al.Journal of Cell Science|February 24, 2006
Disruption of G1-phase phospholipid turnover by inhibition of Ca2+-independent phospholipase A2 induces a p53-dependent cell-cycle arrest in G1 phaseXu Hannah Zhang, Chunying Zhao, Konstantin Seleznev, et al.Endocrinology|February 28, 2009
Advanced glycation end products inhibit glucose-stimulated insulin secretion through nitric oxide-dependent inhibition of cytochrome c oxidase and adenosine triphosphate synthesisZhengshan Zhao, Chunying Zhao, Xu Hannah Zhang, et al.Pageof 2