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Inflammatory Bowel Diseases|October 9, 2018
Death-Domain-Receptor 3 Deletion Normalizes Inflammatory Gene Expression and Prevents Ileitis in Experimental Crohn's DiseaseLudovica F Buttó, Li-Guo Jia, Kristen O Arseneau, et al.
American Journal of Physiology. Gastrointestinal and Liver Physiology|August 18, 2018
Absence of leptin signaling allows fat accretion in cystic fibrosis miceIlya R Bederman, Gavriella Pora, Maureen O'Reilly, et al.
The American Journal of Pathology|February 25, 2016
IL-33 Drives Eosinophil Infiltration and Pathogenic Type 2 Helper T-Cell Immune Responses Leading to Chronic Experimental IleitisCarlo De Salvo, Xiao-Ming Wang, Luca Pastorelli, et al.
Cellular and Molecular Gastroenterology and Hepatology|August 23, 2024
Quantifying Forms and Functions of Enterohepatic Bile Acid Pools in MiceKoichi Sudo, Amber Delmas-Eliason, Shannon Soucy, et al.
Nature|April 8, 2021
CAR directs T cell adaptation to bile acids in the small intestineMei Lan Chen, Xiangsheng Huang, Hongtao Wang, et al.
Immunity|December 21, 2017
The Xenobiotic Transporter Mdr1 Enforces T Cell Homeostasis in the Presence of Intestinal Bile AcidsWei Cao, Hisako Kayama, Mei Lan Chen, et al.
Biorxiv : the Preprint Server for Biology|January 23, 2024
Clonal <i>Parabacteroides</i> from Gut Microfistulous Tracts as Transmissible Cytotoxic Succinate-Commensal Model of Crohn's Disease ComplicationsVaidhvi Singh, Gail West, Claudio Fiocchi, et al.
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